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  • Articles: DFG German National Licenses  (2)
  • 1985-1989  (2)
  • Perfusion systems TBW (percent of body weight) rose significantly  (1)
  • Radium-226  (1)
Source
  • Articles: DFG German National Licenses  (2)
Material
Years
  • 1985-1989  (2)
Year
Keywords
  • 1
    Electronic Resource
    Electronic Resource
    An experimental study of the uptake and loss of Ra-226 by the tissue of the tropical freshwater musselVelesunio angasi (Sowerby) under varying Ca and Mg water concentrations (1986)
    Springer
    Hydrobiologia 139 (1986), S. 59-80 
    Details
    ISSN: 1573-5117
    Keywords: Radium-226 ; uptake ; mussel ; calcium ; magnesium ; metabolic analogue ; competitive inhibition ; uranium ; mining
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Abstract Freshwater musselsVelesunio angasi (Sowerby) from Magela Creek, Alligator Rivers Region, Northern Territory, Australia, experimentally exposed to mean elevated Ra-226 water concentrations ranging between 0.95 and 1.85 Bq 1−1 for 28 days, accumulated Ra-226 in their tissue to mean concentrations ranging from 2.8 to 4.8 Bq per gram of dry tissue. The Ra-226 (log10) was accumulated in a linear pattern over exposure periods of 28 and 56 days. Mussel size and sex had little or no effect on the rates of uptake of Ra-226 per gram of tissue. Increased Ca and Mg water concentrations, both in combination and singly, reduced the rate of uptake of Ra-226 by mussel tissue. The experimental data are consistent with Ra-226 accumulation being inversely proportional to both Ca and Mg water concentrations; for Ca the constant of proportionality i.e. $$Ra = \frac{C}{{[Ca]}}$$ is unity; for Mg it is about 0.1. The results indicate competitive inhibition of the uptake of Ra-226 by Ca, i.e. that the mussel treated Ra-226 as a metabolic analogue of Ca; however, there are other possible interpretations of these results that need not invoke competitive inhibition. For Mg the results suggest involvement of some other mechanism(s) apart from or in addition to competitive inhibition of Ra-226 by Mg. Exposure of mussels that had accumulated Ra-226 under field and laboratory conditions to radium-free water for up to 286 days resulted in no significant loss (P 〉 0.05) of Ra-226 from the tissue. This indicates a very long biological half-life for Ra-226 in the tissue ofV. angasi.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00770242
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Body fluid compartment changes following cardiopulmonary bypass in dogs (1988)
    Springer
    Pediatric surgery international 3 (1988), S. 351-353 
    Details
    ISSN: 1437-9813
    Keywords: Body fluid compartments ; Cardiopulmonary bypass ; Perfusion systems TBW (percent of body weight) rose significantly
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract To investigate whether pulsatile perfusion affects postoperative edema, we examined weight, total body water (TBW), and extracellular fluid (ECF) following cardiopulmonary bypass (CPB) in dogs using three different perfusion systems. Fourteen dogs were divided into three groups differing only in the perfusion system used: Sarns nonpulsatile and pulsatile roller pumps and a University of Texas pulsatile pump. Weight, TBW, and ECF were measured preoperatively and on postoperative days (POD) 1, 3, and 7. No significant differences were seen in body weight, TBW, or ECF between groups. Body TBW (percent of body weight) rose significantly (P=0.005) to 72% on POD 3 and 7. ECF (percent of body weight) rose to 58% by POD 7 (P=0.008). These three perfusion systems produced no differences in the pattern of postoperative body fluid distribution after CPB, suggesting that there is no advantage to pulsatile perfusion for the purpose of decreasing postoperative edema.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00189175
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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