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  • Electronic Resource  (11)
  • 1995-1999  (11)
  • 1
    Electronic Resource
    Electronic Resource
    Semiochemical Parsimony in the Arthropoda (1996)
    Palo Alto, Calif. : Annual Reviews
    Annual Review of Entomology 41 (1996), S. 353-374 
    Details
    ISSN: 0066-4170
    Source: Annual Reviews Electronic Back Volume Collection 1932-2001ff
    Topics: Biology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1146/annurev.en.41.010196.002033
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  • 2
    Electronic Resource
    Electronic Resource
    Reviews of current literature (1995)
    Springer
    International urogynecology journal 6 (1995), S. 119-122 
    Details
    ISSN: 1433-3023
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01962586
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Rezidivierender Bindehauttumor (1997)
    Springer
    Der Ophthalmologe 94 (1997), S. 752-753 
    Details
    ISSN: 1433-0423
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s003470050199
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  • 4
    Electronic Resource
    Electronic Resource
    Sekundärglaukom durch Iristumor (1999)
    Springer
    Der Ophthalmologe 96 (1999), S. 342-343 
    Details
    ISSN: 1433-0423
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s003470050416
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    Paper (German National Licenses)
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  • 5
    Electronic Resource
    Electronic Resource
    Einseitig rotes Auge und erhöhter intraokularer Druck (1997)
    Springer
    Der Ophthalmologe 94 (1997), S. 604-605 
    Details
    ISSN: 1433-0423
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s003470050168
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    Paper (German National Licenses)
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  • 6
    Electronic Resource
    Electronic Resource
    Nitric oxide in ischaemic acute renal failure of streptozotocin diabetic rats (1996)
    Springer
    Diabetologia 39 (1996), S. 1036-1040 
    Details
    ISSN: 1432-0428
    Keywords: Keywords Nitric oxide ; nitrite ; nitrate ; l-arginine ; N-omega-nitro-l-arginine ; ischaemic acute renal failure ; diabetes mellitus ; streptozotocin ; rats.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Changes in nitric oxide (NO) levels were determined in ischaemic acute renal failure in streptozotocin-induced diabetes mellitus rats. Two weeks after streptozotocin administration and immediately after right nephrectomy, the left renal artery was occluded for 60 min. Similar procedures were carried out in non-diabetic rats. The nitrite (NO2) + nitrate (NO3) levels were measured in plasma and urine. The effects of chronic oral supplementation with l-arginine and an NO synthase inhibitor (N-omega-nitro-l-arginine) were also studied in both diabetic and non-diabetic rats before and after renal artery clamping. The rats with diabetic acute renal failure had a much lower creatinine clearance (90 ± 22 μl · min−1· 100 g body weight −1, p 〈 0.005), and higher fractional excretion of sodium (FENa)% (10.90 ± 4.2, p 〈 0.001) and protein excretion (2078 ± 69 μg/ml creatinine clearance, p 〈 0.001) compared with the respective values in the non-diabetic groups (163 ± 30; 1.46 ± 86; 453.3 ± 31). The plasma and urine NO2 + NO3 levels were significantly higher in the untreated diabetic rats compared with the untreated normal rats before ischaemia (p 〈 0.001). The ischaemic acute renal failure in non-diabetic rats increased the plasma and urinary NO2 + NO3 excretion after ischaemia. The urinary excretion of these metabolites decreased significantly and their plasma levels remained unchanged in the ischaemic diabetic rats. The l-arginine administration resulted in a small but significantly higher creatinine clearance after clamping in the non-diabetic rats. The NO synthase inhibitor caused deterioration in renal function in all ischaemic and non-ischaemic groups. In summary, the greater vulnerability to ischaemia of the diabetic kidney seems to be associated with both impaired response to and impaired production of NO. [Diabetologia (1996) 39: 1036–1040]
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00400651
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  • 7
    Electronic Resource
    Electronic Resource
    Nitric oxide in ischaemic acute renal failure of streptozotocin diabetic rats (1996)
    Springer
    Diabetologia 39 (1996), S. 1036-1040 
    Details
    ISSN: 1432-0428
    Keywords: Nitric oxide ; nitrite ; nitrate ; l-arginine ; N-omega-nitro-l-arginine ; ischaemic acute renal failure ; diabetes mellitus ; streptozotocin ; rats
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Changes in nitric oxide (NO) levels were determined in ischaemic acute renal failure in streptozotocin-induced diabetes mellitus rats. Two weeks after streptozotocin administration and immediately after right nephrectomy, the left renal artery was occluded for 60 min. Similar procedures were carried out in non-diabetic rats. The nitrite (NO2) + nitrate (NO3) levels were measured in plasma and urine. The effects of chronic oral supplementation with l-arginine and an NO synthase inhibitor (N-omega-nitro-l-arginine) were also studied in both diabetic and non-diabetic rats before and after renal artery clamping. The rats with diabetic acute renal failure had a much lower creatinine clearance (90±22 Μl · min−1 · 100 g body weight−1, p〈0.005), and higher fractional excretion of sodium (FENa)% (10.90±4.2, p〈0.001) and protein excretion (2078±69 Μg/ml creatinine clearance, p〈0.001) compared with the respective values in the non-diabetic groups (163±30; 1.46±86; 453.3±31). The plasma and urine NO2 + NO3 levels were significantly higher in the untreated diabetic rats compared with the untreated normal rats before ischaemia (p〈0.001). The ischaemic acute renal failure in non-diabetic rats increased the plasma and urinary NO2 + NO3 excretion after ischaemia. The urinary excretion of these metabolites decreased significantly and their plasma levels remained unchanged in the ischaemic diabetic rats. The l-arginine administration resulted in a small but significantly higher creatinine clearance after clamping in the non-diabetic rats. The NO synthase inhibitor caused deterioration in renal function in all ischaemic and non-ischaemic groups. In summary, the greater vulnerability to ischaemia of the diabetic kidney seems to be associated with both impaired response to and impaired production of NO.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00400651
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    Paper (German National Licenses)
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  • 8
    Electronic Resource
    Electronic Resource
    Elucidation of Dimethylalkylpyrazines from the Ant Streblognathus aethiopicus by GC-FTIR (1998)
    Springer
    Journal of chemical ecology 24 (1998), S. 125-134 
    Details
    ISSN: 1573-1561
    Keywords: Pyrazines ; GC-FTIR ; Streblognathus ; gamergate
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology
    Notes: Abstract The presumed mandibular gland secretions from the workers and gamergates of two colonies of Streblognathus aethiopicus have been found to contain a homologous series of 3,5-dimethyl-2-alkylpyrazines (3,5-dimethyl-2-butylpyrazine, 1; 3,5-dimethyl-2-pentylpyrazine, 2; and 3,5-dimethyl-2-hexylpyrazine, 3). The structures of these compounds were determined from their mass spectra along with a comparison of their GC-FTIR spectra with those of all the isomers of a lower homolog. This is the first time that this combination of techniques has been applied to the study of insect pyrazines. The relative amounts and proportions of these compounds were different for gamergates and workers, with the latter containing 2–10 times more total pyrazines and having a higher proportion of 2 and 3. The possible significance of this finding is discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/A:1022345114057
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    Paper (German National Licenses)
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  • 9
    Electronic Resource
    Electronic Resource
    Noninvasive measurement of the Bayliss effect in retinal autoregulation (1999)
    Springer
    Graefe's archive for clinical and experimental ophthalmology 237 (1999), S. 296-300 
    Details
    ISSN: 1435-702X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract · Purpose: The Bayliss effect describes the reaction of smooth muscle cells in the arterial wall to changes in blood pressure. A rise in mean arterial blood pressure (MAP) causes an autoregulatory myogenic vessel constriction by smooth muscle cells in the arterial wall. The responsiveness of retinal vessels to changes in MAP were analyzed using the Retinal Vessel Analyzer (RVA). · Methods: Continuous measurement of retinal arterial vessels was performed in 40 healthy volunteers (age 18–56 years.) over a 9-min period. After a 3-min baseline measurement (phase I), isometric exercise caused a rise in MAP over the next 3 min (phase II). During the last 3 min (phase III) recovery was observed. Blood pressure and ECG were documented simultaneously throughout the experiment. · Results: Exercise caused a significant rise of 22.8 (±6.0) mmHg in MAP (phase II vs phase I: P〈0.001). Retinal arterioles showed 5.5% (±2.8%) vasoconstriction (P〈0.001). During phase III vessel diameters returned to normal, with no difference from phase I (P=0.179). · Conclusion: Noninvasive measurement and quantitative analysis of the Bayliss effect in human retinal vessels by means of the RVA is possible. Analysis of retinal arterial autoregulation may provide valuable insight into pathologic conditions such as diabetic or hypertensive retinopathy.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s004170050236
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  • 10
    Electronic Resource
    Electronic Resource
    The mouse goosecoïd gene (Gsc) maps to the telomeric part of mouse Chromosome 12 (1995)
    Springer
    Mammalian genome 6 (1995), S. 816-817 
    Details
    ISSN: 1432-1777
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00539011
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