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  • 1
    Electronic Resource
    Electronic Resource
    Thermally induced structural modification in Pt/C X-ray multilayer mirrors fabricated by electron beam evaporation (1996)
    Springer
    Applied physics 62 (1996), S. 29-32 
    Details
    ISSN: 1432-0630
    Keywords: 68.35.Fx ; 68.60.Dv ; 68.65. + g
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics , Physics
    Notes: Abstract Platinum-carbon multilayer mirrors with a bilayer spacing of 50 Å were fabricated in an ultrahigh vacuum electron beam evaporator. The thermal stability of these multilayers was studied under vacuum annealing using X-ray reflectivity and X-ray diffraction. Up to 450°C, the bilayer spacing increases monotonically accompanied by a gradual increase in crystallite size and grain texture. At 500°C multilayer reflection vanishes, platinum crystallites grow abruptly, and there is a strong texture of platinum in the [220] -plane. Possible reasons for thermally induced structural modifications in these multilayers are discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01568083
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Evidence that glucagon stimulates insulin secretion through its own receptor in rats (1995)
    Springer
    Diabetologia 38 (1995), S. 274-276 
    Details
    ISSN: 1432-0428
    Keywords: Key words Glucagon ; insulin secretion ; exendin (9 ; 39) ; GLP-1 ; pancreas perfusion.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Since glucagon-like peptide-1 (7–36) amide (7–37) (GLP-1) has been found to be a potent insulinotropic hormone, it has been postulated that glucagon stimulates insulin secretion from islet beta cells through the GLP-1 receptor. We therefore examined the effects of a GLP-1 receptor antagonist, exendin (9–39) amide, on glucagon- or GLP-1-stimulated insulin release from isolated perfused rat pancreas. When infusion of 100 nmol/l exendin (9–39) amide was started 5 min before that of 1 nmol/l glucagon, the stimulation of insulin release by glucagon was similar to that found in the control situation (preinfusion with vehicle alone). By contrast, when 0.3 nmol/l GLP-1 was used in the same experimental setting, exendin (9–39) amide clearly inhibited insulin release. These results indicate that glucagon stimulates insulin release mainly through glucagon receptors but not GLP-1 receptors on islet beta cells. [Diabetologia (1995) 38: 274–276]
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00400630
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    Paper (German National Licenses)
    Fulltext
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