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  • 2000-2004  (1)
  • 1990-1994  (2)
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  • 1
    Electronic Resource
    Electronic Resource
    Oxford, UK : Blackwell Publishing Ltd
    Plant pathology 41 (1992), S. 0 
    ISSN: 1365-3059
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Agriculture, Forestry, Horticulture, Fishery, Domestic Science, Nutrition
    Notes: The epidemic development of Ascochyta fabae in artificially infected field trials was studied in order to define resistance criteria and assess the variation in resistance of infected lines of faba bean. Large differences were demonstrated among French winter lines during the 1985/86 and 1986/87 growing seasons. All the scoring systems allowed the differentiation of very susceptible and resistant material, but disease indices, which account for the number and intensity of lesions, were more precise. The different phases of the epidemic are described on the basis of the variation in disease incidence on foliage and on pods during the 1986/87 cropping season in Brittany. There were three phases in disease development: initiation, spread throughout foliage, and pod infection, and these were all influenced by host resistance. On a very susceptible line, the disease spread was seen as an increase in the number and size of lesions and rapidly developed up the plant. Low disease indices observed on the resistant line 29 H were due to the small size and low frequency of lesions, resulting in a reduced area of foliage affected.
    Type of Medium: Electronic Resource
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  • 2
    Electronic Resource
    Electronic Resource
    Oxford, UK : Blackwell Publishing Ltd
    Plant pathology 43 (1994), S. 0 
    ISSN: 1365-3059
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Agriculture, Forestry, Horticulture, Fishery, Domestic Science, Nutrition
    Notes: The production of ascochitine by seven isolates of Ascochyta fabae accounted for the toxicity of culture filtrates of the fungus to cells isolated from leaves of Vicia faba. The LD50 value for cells from cultivars that were susceptible, tolerant or resistant to the fungus was similar i.e. 3·0 × 10−5m, 3·8 × 10−5m and 2·9 × 10−5 M, respectively. Ascochitine affected neither the germination of seeds nor the growth of mature plants at 5·17 × 10−4m but caused necrosis and wilting of plant cuttings at 2·5 × 10−4m and 5·10−4m. There was no association between virulence of 16 isolates of A. fabae for three cultivars of V.faba and the production of ascochitine in vitro. One isolate produced no ascochitine in vitro and yet was the most virulent for two of three cultivars. The toxin could not be extracted from infected plants.
    Type of Medium: Electronic Resource
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  • 3
    Electronic Resource
    Electronic Resource
    Oxford, U.K. and Cambridge, USA : Blackwell Science Ltd
    Plant pathology 49 (2000), S. 0 
    ISSN: 1365-3059
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Agriculture, Forestry, Horticulture, Fishery, Domestic Science, Nutrition
    Notes: The effect of epidemics of Mycosphaerella pinodes on crop growth, radiation interception efficiency (RIE) and radiation use efficiency (RUE) was studied in field conditions and the growth of diseased crop was modelled. Natural epidemics were simulated in field plots by dispersion on the ground of barley grains colonized by the fungus. Growth and leaf area index (LAI) were measured in healthy and diseased plots during the growth season. Epidemics affected crop growth mainly by decreasing RUE, with a slight decrease in RIE. This was probably due to the strong effect of M. pinodes on leaf photosynthesis and the lateness of Mycosphaerella blight epidemics on spring pea, which became serious when the canopy was already formed, thus decreasing RIE only by accelerating the senescence of leaves. The data were used to evaluate the performance of a crop growth model for diseased pea crops. The model comprised the decrease in photosynthesis rate in the leaves, the vertical gradient of disease intensity and the differences in photosynthetic function of the various layers of the canopy. This model, validated over 2 years in the field, accurately simulated the crop growth in diseased plots. Thus the decrease in RUE may be accounted for solely by photosynthesis losses in diseased leaves. This simple model may be used for disease management, defining damage thresholds for chemical application and criteria for tolerant variety selection.
    Type of Medium: Electronic Resource
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