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  • 2000-2004  (1)
  • 1975-1979  (1)
  • allozymes  (1)
  • microflora  (1)
  • 1
    Electronic Resource
    Electronic Resource
    An acid phosphatase locus expressed in mouse kidney (Apk) and its genetic location on chromosome 10 (1978)
    Springer
    Biochemical genetics 16 (1978), S. 239-245 
    Details
    ISSN: 1573-4927
    Keywords: mouse ; acid phosphatase ; Apk, chromosome 10 ; lysosomal enzymes ; isozymes ; allozymes
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology
    Notes: Abstract A genetic locus controlling the electrophoretic mobility of an acid phosphatase in mouse kidney is described. This locus, called acid phosphatase-kidney (Apk), is not expressed in erythrocytes, liver, spleen, heart, lung, brain, skeletal muscle, stomach, or testes. The product of Apk hydrolyzes the substrate naphthol AS-MX phosphoric acid but is not active on a-naphthylphosphate or 4-methylumbelliferylphosphate. It is not inactivated by 50 C for 1 hr, nor is its electrophoretic mobility altered by incubation with neuraminidase. The locus is invariant among 31 inbred strains (Apk a), with a variant allele (Apk m) observed only in Mus musculus molossinus. Codominant expression was observed in F1 hybrids of M. m. molossinus and inbred strains. Apk was mapped on Chr 10, near the neurological mutant waltzer (v).
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00484081
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    In Vitro Metabolism of Fumonisin B1 by Ruminal Microflora (2000)
    Springer
    Veterinary research communications 24 (2000), S. 379-387 
    Details
    ISSN: 1573-7446
    Keywords: biodegradation ; fumonisin B1 ; metabolites ; microflora ; mycotoxin ; rumen ; short-chain fatty acid
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Fumonisin B1 (FB1) is a mycotoxin produced by Fusarium moniliforme and F. proliferatum. Little is known of its metabolic fate after oral ingestion in ruminants, but these animals are reported to be tolerant towards FB1. The metabolism of this mycotoxin was evaluated following incubation (1 μg/ml) in ruminal fluid for up to 72 h, in the presence or absence of alfalfa as a substrate for microbial growth, using a model rumen (sealed flask, anaerobic conditions, exclusion of light, gentle agitation, 39°C). The decrease in FB1 concentration and the production of short-chain fatty acids were determined. FB1 had no effect on SCFA production. After 72 h incubation, FB1 depletion was 12% and 18% in samples with and without alfalfa, respectively. No hydrolysed metabolites (aminopolyols or aminopentol) were detected. These results indicate that FB1 is poorly metabolized in the rumen and suggest that such metabolism is not the cause of the tolerance to this toxin displayed by ruminants.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/A:1006422200226
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    Paper (German National Licenses)
    Fulltext
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