Library

X
feed icon rss

Your email was sent successfully. Check your inbox.

An error occurred while sending the email. Please try again.

Proceed reservation?

Export
Filter
  • 1995-1999  (2)
  • 1985-1989  (3)
  • 1
    Electronic Resource
    Electronic Resource
    Rotational rainbow scattering with large Δ j: Energy dependence of the anisotropy of the Na2–Ne, Ar interaction potential (1987)
    College Park, Md. : American Institute of Physics (AIP)
    The Journal of Chemical Physics 86 (1987), S. 2680-2684 
    Details
    ISSN: 1089-7690
    Source: AIP Digital Archive
    Topics: Physics , Chemistry and Pharmacology
    Notes: Differential cross sections for rotational transitions in Na2–Ne, Ar collisions are measured up to very large Δj. It is shown that the energy dependent anisotropy ΔR(E) of the interaction potential can be determined from these data using simple classical relations of ΔR and the rainbow angle θR. Excellent agreement with ab initio data is demonstrated and underlines the usefulness of this concept.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.452070
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Supernumerary rotational rainbows in Na2–He, Ne, Ar scattering (1987)
    College Park, Md. : American Institute of Physics (AIP)
    The Journal of Chemical Physics 86 (1987), S. 2685-2688 
    Details
    ISSN: 1089-7690
    Source: AIP Digital Archive
    Topics: Physics , Chemistry and Pharmacology
    Notes: We report the resolution of supernumerary rotational rainbows for rotationally inelastic ji → jf=0 transitions in Na2–He, Ne, Ar scattering. The relation of the angular position of the supernumerary maxima and the anisotropy ΔR of the interaction potential is established. The analysis yields the energy dependent anisotropy ΔR(E) in very good agreement with data from other sources.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.452071
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Angularly resolved vibrational excitation in Na2–He collisions (1986)
    College Park, Md. : American Institute of Physics (AIP)
    The Journal of Chemical Physics 84 (1986), S. 756-763 
    Details
    ISSN: 1089-7690
    Source: AIP Digital Archive
    Topics: Physics , Chemistry and Pharmacology
    Notes: We report angle-resolved measurements of vi=0 → vf=1 vibrational transitions in Na2–He collisions at an energy of 90 meV. The agreement with calculated cross sections using an ab initio surface is good, both in the angular variation of the cross section as well as with respect to its magnitude relative to the vibrationally elastic process. The calculated (vi=0, ji=0) → (vf=1, jf ) differential cross sections are discussed in some more detail. They show structure, in addition to the rainbow oscillations, related to the fact that the vibrational transition probability vanishes for a specific approach angle.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.450573
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 4
    Electronic Resource
    Electronic Resource
    Electrocardiological profile and proarrhythmic effects of quinidine, verapamil and their combination: a mapping study (1995)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 352 (1995), S. 94-101 
    Details
    ISSN: 1432-1912
    Keywords: Quinidine ; Verapamil ; Proarrhythmic risk ; Epicardial mapping ; Arrhythmia Atrioventricular conduction
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Quinidine and verapamil are widely used as antiarrhythmic agents and their combination is often used in the treatment of supraventricular tachycardia. This study was undertaken to clarify, whether these drugs exert proarrhythmic effects on the ventricles in therapeutic concentrations and whether possible arrhythmogenic effects might be enhanced by combination. Isolated rabbit hearts perfused according to the Langendorff technique were treated with increasing concentrations of quinidine (0.05 to 3.5 μM) or verapamil (5 to 50 μM) or of their combination (70:1 or 10:1; quinidine:verapamil) corresponding to common low, medium and high free therapeutic concentrations. The epicardial activation process was measured using a computer assisted mapping system for unipolar multichannel recording (256 channels simultaneously). Both substances prolonged the atrioventricular conduction time PQ. This effect was even more pronounced if the 70:1 combination was administered. The activation pattern was altered by both drugs and their combination to the same extent as became obvious from analysis of local activation vectors and of localisation of breakthroughpoints of epicardial activation for heart beats under control conditions and under drug treatment. The epicardial potential durations were prolonged by quinidine and to the same degree by the combinations, but not by verapamil alone. The total activation time was prolonged under the influence of quinidine and if the 70:1 combination was given. Both substances exerted a negative inotropic effect which was enhanced in an additive manner if both drugs were combined. In parallel the coronary flow was diminished. From these results it is concluded that (1) in this therapeutic concentration range quinidine possess a greater proarrhythmic risk than verapamil, (2) that both drugs' PQ prolonging effect can be enhanced by combination, (3) that combination does not enhance the proarrhythmic effects but the negative inotropic effects.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00169195
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 5
    Electronic Resource
    Electronic Resource
    Electrocardiological profile and proarrhythmic effects of quinidine, verapamil and their combination: a mapping study (1995)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 352 (1995), S. 94-101 
    Details
    ISSN: 1432-1912
    Keywords: Key words Quinidine ; Verapamil ; Proarrhythmic ; risk ; Epicardial mapping ; Arrhythmia ; Atrioventricular conduction
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract  Quinidine and verapamil are widely used as antiarrhythmic agents and their combination is often used in the treatment of supraventricular tachycardia. This study was undertaken to clarify, whether these drugs exert proarrhythmic effects on the ventricles in therapeutic concentrations and whether possible arrhythmogenic effects might be enhanced by combination. Isolated rabbit hearts perfused according to the Langendorff technique were treated with increasing concentrations of quinidine (0.05 to 3.5 μM) or verapamil (5 to 50 nM) or of their combination (70:1 or 10:1; quinidine:verapamil) corresponding to common low, medium and high free therapeutic concentrations. The epicardial activation process was measured using a computer assisted mapping system for unipolar multichannel recording (256 channels simultaneously). Both substances prolonged the atrioventricular conduction time PQ. This effect was even more pronounced if the 70:1 combination was administered. The activation pattern was altered by both drugs and their combination to the same extent as became obvious from analysis of local activation vectors and of localisation of breakthroughpoints of epicardial activation for heart beats under control conditions and under drug treatment. The epicardial potential durations were prolonged by quinidine and to the same degree by the combinations, but not by verapamil alone. The total activation time was prolonged under the influence of quinidine and if the 70:1 combination was given. Both substances exerted a negative inotropic effect which was enhanced in an additive manner if both drugs were combined. In parallel the coronary flow was diminished. From these results it is concluded that (1) in this therapeutic concentration range quinidine possess a greater proarrhythmic risk than verapamil, (2) that both drugs’ PQ prolonging effect can be enhanced by combination, (3) that combination does not enhance the proarrhythmic effects but the negative inotropic effects.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00169195
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
Close ⊗
This website uses cookies and the analysis tool Matomo. More information can be found here...