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  • 1
    Electronic Resource
    Electronic Resource
    First principles theory of spin waves in Fe, Co, and Ni (1999)
    [S.l.] : American Institute of Physics (AIP)
    Journal of Applied Physics 85 (1999), S. 4830-4832 
    Details
    ISSN: 1089-7550
    Source: AIP Digital Archive
    Topics: Physics
    Notes: Using constrained local spin density functional theory it is possible to directly calculate the adiabatic magnon dispersion curve. Small amplitude magnons with wavelengths between 2 and 32 atomic layers were constructed and the energy change with respect to amplitude within the frozen potential approximation was calculated. The resulting dispersion curves give the spin wave stiffness for [111] face-centered-cubic Co and Ni in fair agreement with experiment. In Fe the stiffness was calculated along [100] and [110] directions. The two values of stiffness were found to be nearly equal to each other and roughly half the measured value. It was found that the calculated Fe stiffness did not change upon addition of gradient corrections to the local density approximation, indicating that the source of discrepancy is not likely a failure of the local density approximation. It is argued that the adiabatic approximation is more likely the source of discrepancy since the magnetic interactions in Fe are longer ranged than those in Co or Ni, thus making the dynamical retardation effect more important in Fe. © 1999 American Institute of Physics.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.370035
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  • 2
    Electronic Resource
    Electronic Resource
    Calculation of the canting angle dependence of the resistivity in Cu|Co spin valves : The 41st annual conference on magnetism and magnetic materials (1997)
    [S.l.] : American Institute of Physics (AIP)
    Journal of Applied Physics 81 (1997), S. 4008-4010 
    Details
    ISSN: 1089-7550
    Source: AIP Digital Archive
    Topics: Physics
    Notes: We consider the in-plane electrical conductivity of a spin valve, specifically, a system consisting of a copper slab between two cobalt slabs. We calculate the dependence of the resistivity on the canting angle, where the canting angle θ is defined as the angle between the magnetization vectors of two ferromagnetic slabs in a spin valve. To calculate the electronic structure, we utilize the layer-KKR formalism. Electron scattering by impurities, phonons, magnons, etc. is modeled using a layer and spin-dependent complex self-energy. Scattering rates are chosen to match Cu and Co resistivities. We assume a spin asymmetry scattering rate factor of 7 in Co, matching the Fermi-level minority-to-majority density-of-states ratio. No additional interfacial scattering is included. The nonlocal layer dependent conductivity is calculated using the Kubo–Greenwood formula for systems consisting of 3 and 7 monolayer fcc (111) Cu slabs in Co. We find electron channeling in Cu dominates the conductivity and at θ=π/2 the resistivity is increased by 4.9% and 2.3% from a linear 1−cos θ dependence. We find giant magnetoresistive GMR values of 64% and 36% for 3 and 7 monolayers of Cu, respectively. © 1997 American Institute of Physics.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.364922
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  • 3
    Electronic Resource
    Electronic Resource
    Lack of C4 photosynthetic metabolism in ears of C3 cereals (1995)
    Oxford, UK : Blackwell Publishing Ltd
    Plant, cell & environment 18 (1995), S. 0 
    Details
    ISSN: 1365-3040
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology
    Notes: There is continuing controversy over whether a degree of C4 photosynthetic metabolism exists in ears of C3 cereals. In this context, CO2 exchange and the initial products of photosynthesis were examined in flag leaf blades and various ear parts of two durum wheat (Triticum durum Desf.) and two six-rowed barley (Hordeum vulgare L.) cultivars. Three weeks after anthesis, the CO2 compensation concentration at 210 mmol mol−1 O2 in durum wheat and barley ear parts was similar to or greater than that in flag leaves. The O2 dependence of the CO2 compensation concentration in durum wheat ear parts, as well as in the flag leaf blade, was linear, as expected for C3 photosynthesis. In a complementary experiment, intact and attached ears and flag leaf blades of barley and durum wheat were radio-labelled with 14CO2 during a 10s pulse, and the initial products of fixation were studied in various parts of the ears (awns, glumes, inner bracts and grains) and in the flag leaf blade. All tissues assimilated CO2 mainly by the Calvin (C3) cycle, with little fixation of 14CO2 into the C4 acids malate and aspartate (about 10% or less). These collective data support the conclusion that in the ear parts of these C3 cereals C4 photosynthetic metabolism is nil.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-3040.1995.tb00571.x
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  • 4
    Electronic Resource
    Electronic Resource
    Genetic fine mapping of the Miyoshi myopathy locus and exclusion of eight candidate genes (1998)
    Springer
    Neurogenetics 1 (1998), S. 189-196 
    Details
    ISSN: 1364-6753
    Keywords: Key words Miyoshi myopathy ; Genetic fine mapping ; Candidate genes
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: ABSTRACT Miyoshi myopathy (MM) is an early adult-onset, autosomal recessive disorder characterized by weakness and muscular atrophy starting in the distal muscles. The disease locus has been previously mapped by linkage analysis to chromsome 2p using the microsatellite marker D2S291. Initial haplotype analysis of markers in families from three different origins (North American, Japanese, and Tunisian) suggested that the MM gene is located in a 4-cM region flanked by markers D2S292 on the telomeric side and D2S286 on the centromeric side. To delineate critical recombination events revealing a more refined localization of the MM gene, we have determined the pattern of segregation of 12 marker loci in two consanguineous families of Tunisian origin. In this study we have: (1) detected recombination events with the disease locus in one family, placing the MM gene most likely between markers D2S443 (CHLC.GGAA4D07.1876) and D2S2109; (2) generated a yeast artificial chromosome contig that spans approximately 3.8 megabases and extends from marker D2S358 to marker D2S286; (3) physically mapped 21 polymorphic markers, 5 genes, 3 STSs, and 1 EST within this contig; (4) detected and mapped a new polymorphism within this interval, allowing us to further reduce the MM locus to a 360-kilobase segment; (5) mapped the gene for the cytoskeletal protein β-adducin within the MM candidate region, failing to find a consistent pattern of mutation of this gene in our MM patients; (6) excluded seven other candidate myopathy genes from the Miyoshi locus.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s100480050028
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  • 5
    Electronic Resource
    Electronic Resource
    Identification of two mutations and a polymorphism in the chloride channel CLCN-1 in patients with Becker's generalized myotonia (1998)
    Springer
    Neurogenetics 1 (1998), S. 185-188 
    Details
    ISSN: 1364-6753
    Keywords: Key words Myotonia congenita ; Thomsen's disease ; Becker's generalized myotonia ; Muscle chloride channel ; CLCN-1 gene
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: ABSTRACT Myotonia congenita is an inherited muscle disorder characterized by muscle stiffness and hypertrophy. Its clinical phenotype depends, in part, on whether it is inherited as a dominant or recessive trait, respectively designated Thomsen's disease or Becker's generalized myotonia (BGM). In either case, it is associated with abnormalities in the muscle currents that are linked to the gene (CLCN-1) on human chromosome 7q35 encoding the skeletal muscle chloride channel. Single-strand conformation polymorphism analysis was used to screen two families with the BGM for mutations in the CLCN-1 gene. Two new mutations were found (G 201ins and A317Q). The latter mutation has been previously described in Thomsen's disease.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s100480050027
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