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  • 1990-1994  (6)
  • 1925-1929
  • 1900-1904
  • 1880-1889
  • Electrolyte-induced demyelination  (4)
  • Colorectal surgery  (2)
  • 1
    Electronic Resource
    Electronic Resource
    Advantages of handsewn over stapled bowel anastomosis (1991)
    Springer
    Diseases of the colon & rectum 34 (1991), S. 442-448 
    Details
    ISSN: 1530-0358
    Keywords: Colorectal surgery ; Suture techniques ; Surgical anastomosis ; Surgical staplers ; Dogs
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Bowel anastomoses are conventionally performed using a handsewn technique or a stapling device. Each has potential benefits and disadvantages. The most clinically significant complications of the bowel anastomosis are anastomotic leakage and stricture formation. The indices of healing and tissue cohesion were compared dynamically over time in 24 dogs randomized to undergo either a standard two-layer handsewn anastomosis or a stapled anastomosis with the Premium CEEATM (United States Surgical Corporation, Norwalk, CT). Animals were sacrificed at 1, 4, 7, and 28 days postoperatively. Each anastomosis was evaluated for anastomotic index, burst pressure, collagen content, and histologic appearance. The anastomotic index was similar on postoperative day (POD) 1, 4, and 7; but on day 28 all handsewn anastomoses had larger diameters than the widest CEEATM anastomosis. Burst pressure was higher in handsewn anastomoses at all intervals. Collagen content tended to be higher on POD 7 in the CEEATM anastomoses. Histological evaluation showed more complete epithelialization and less inflammation in handsewn anastomoses on POD 28. The higher level of collagen in the CEEATM anastomoses on POD 7 may be implicated in the tendency toward stricture formation found with this type of anastomosis. This study demonstrates that the greater speed and ease of the stapled anastomosis is offset by the greater strength, reduced tendency to stricture, and more complete healing of the handsewn anastomosis.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02049926
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    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Anastomotic compression button: A new mechanical device for sutureless bowel anastomosis (1990)
    Springer
    Diseases of the colon & rectum 33 (1990), S. 291-297 
    Details
    ISSN: 1530-0358
    Keywords: Surgical staplers ; Colorectal surgery ; Suture techniques ; Laser velocimetry ; Surgical anastomosis ; Dogs
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The anastomotic compression button is a new mechanical device that uses three interlocking polypropylene buttons to produce a sutureless bowel anastomosis. The device is unique in that it allows application of the buttons via a device similar to the popular intraluminal stapler, but it leaves no staples or foreign body of any kind in the bowel wall. The authors compared the 25-mm anastomotic compression button with the 25-mm intraluminal stapler in the colon of dogs. After 28 days, the mucosal blood flow, burst pressure, and anastomotic indices were found to be identical between the anastomotic compression button and the stapler. The anastomotic compression button was easier to use, and microscopic examination showed less ulceration, fibrosis, and inflammation, and better re-epithelialization at the anastomotic compression button site. The anastomotic compression button appears to have the potential to be a superior method compared with stapled anastomoses in the colon.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02055470
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    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Electrolyte-induced demyelination in rats (1994)
    Springer
    Acta neuropathologica 88 (1994), S. 287-292 
    Details
    ISSN: 1432-0533
    Keywords: Blood-brain-barrier ; Central pontine myelinolysis ; Electrolyte-induced demyelination ; Oligodendrocyte ; Pathogenesis
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The blood-brain barrier (BBB) was studied in rats with electrolyte-induced demyelination (EID), an experimental model for central pontine myelinolysis. Intravenously injected peroxidase was extravasated at 3 h post hypertonic saline injection (PHS) into regions frequently involved in EID. Increased pinocytotic activity and focal interendothelial gaps were seen at 3 h PHS and less frequently at 48 h PHS. Measurement of total cerebral water content revealed an increase during the hyponatremic phase. This was followed by a marked increase at 3 h PHS with continued increment at 48 h PHS. Intracellular edema with accumulation of fluid within neurites and astrocytic processes was noted during the hyponatremic phase, whereas extracellular edema developed after hypertonic saline injection. The implications of disrupted BBB and its role in the pathogenesis of EID are discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00310371
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 4
    Electronic Resource
    Electronic Resource
    Electrolyte-induced demyelination in rats (1994)
    Springer
    Acta neuropathologica 88 (1994), S. 287-292 
    Details
    ISSN: 1432-0533
    Keywords: Key words Blood-brain-barrier ; Central pontine ; myelinolysis ; Electrolyte-induced demyelination ; Oligodendrocyte ; Pathogenesis
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The blood-brain barrier (BBB) was studied in rats with electrolyte-induced demyelination (EID), an experimental model for central pontine myelinolysis. Intravenously injected peroxidase was extravasated at 3 h post hypertonic saline injection (PHS) into regions frequently involved in EID. Increased pinocytotic activity and focal interendothelial gaps were seen at 3 h PHS and less frequently at 48 h PHS. Measurement of total cerebral water content revealed an increase during the hyponatremic phase. This was followed by a marked increase at 3 h PHS with continued increment at 48 h PHS. Intracellular edema with accumulation of fluid within neurites and astrocytic processes was noted during the hyponatremic phase, whereas extracellular edema developed after hypertonic saline injection. The implications of disrupted BBB and its role in the pathogenesis of EID are discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00310371
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 5
    Electronic Resource
    Electronic Resource
    Electrolyte-induced demyelination in rats (1994)
    Springer
    Acta neuropathologica 88 (1994), S. 293-299 
    Details
    ISSN: 1432-0533
    Keywords: Blood-brain barrier ; Central pontine myelinolysis ; Electrolyte-induced demyelination ; Oligodendrocyte
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract This study presents the electron microscopic evolution of lesions in electrolyte-induced demyelination (EID) in rats, a lesion which bears striking histological and clinical similarity to central pontine myelinolysis. The earliest change was observed during the hyponatremic phase and consisted of minimal intracellular edema present throughout the brain. Following the injection of hypertonic saline, additional changes were observed which were restricted to sites previously reported to be frequently involved in EID. Early dilatation of the inner tongue of oligodendrocyte cytoplasm in myelinated nerve fibers was observed at 3h post hypertonic saline injection (PHS). This was followed, at 48h PHS, by the appearance of degenerative changes consistent with dying oligodendrocytes. Well-delineated, vacuolar and spongy lesions, seen by light microscopy, were present by 48h PHS at the same sites as above. Electron microscopically, this appearance was found to be due to striking intramyelinic edema. By 96h PHS, macrophages containing myelin and other cellular debris were frequently present at these sites. Concomitantly, myelin sheaths underwent vesicular disruption and disintegration. This sequence of events suggest a lesion of the oligodendrocyte-myelin complex, secondary to initial blood-brain barrier damage and edema.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00310372
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 6
    Electronic Resource
    Electronic Resource
    Electrolyte-induced demyelination in rats (1994)
    Springer
    Acta neuropathologica 88 (1994), S. 293-299 
    Details
    ISSN: 1432-0533
    Keywords: Key words Blood-brain barrier ; Central pontine ; myelinolysis ; Electrolyte-induced demyelination ; Oligodendrocyte
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract This study presents the electron microscopic evolution of lesions in electrolyte-induced demyelination (EID) in rats, a lesion which bears striking histological and clinical similarity to central pontine myelinolysis. The earliest change was observed during the hyponatremic phase and consisted of minimal intracellular edema present throughout the brain. Following the injection of hypertonic saline, additional changes were observed which were restricted to sites previously reported to be frequently involved in EID. Early dilatation of the inner tongue of oligodendrocyte cytoplasm in myelinated nerve fibers was observed at 3   h post hypertonic saline injection (PHS). This was followed, at 48   h PHS, by the appearance of degenerative changes consistent with dying oligodendrocytes. Well-de- lineated, vacuolar and spongy lesions, seen by light microscopy, were present by 48   h PHS at the same sites as above. Electron microscopically, this appearance was found to be due to striking intramyelinic edema. By 96   h PHS, macrophages containing myelin and other cellular debris were frequently present at these sites. Concomitantly, myelin sheaths underwent vesicular disruption and disintegration. This sequence of events suggests a lesion of the oligodendrocyte-myelin complex, secondary to initial blood-brain barrier damage and edema.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00310372
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
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