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Photoluminescence enhancement in post-growth hydrogenated Ga1−xAlxAs (0≤x≤0.32) and GaAs/GaAlAs multilayer structures (1989)

Pavesi, L. ; Martelli, F. ; Martin, D. ; [et al.]

Woodbury, NY : American Institute of Physics (AIP)

Applied Physics Letters 54 (1989), S. 1522-1524

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ISSN: 1077-3118

Source: AIP Digital Archive

Topics: Physics

Notes: Low-temperature photoluminescence measurements in undoped Ga1−xAlxAs (0≤x≤0.32) and GaAs/GaAlAs quantum structures grown by molecular beam epitaxy are performed before and after hydrogen plasma exposure. In both cases we observe a strong enhancement of the luminescence after the exposure. In the GaAlAs epilayers this enhancement clearly depends on the Al concentration in the alloy. The results are explained in terms of passivation of nonradiative traps in the GaAlAs that increases the carriers' lifetime and diffusion length allowing, in the case of the multilayers, a greater number of carriers to recombine in the quantum well.

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1063/1.101339

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GABA-A-mediated gastrin release induced by baclofen in the isolated vascularly perfused rat stomach (1989)

Tacca, M. ; Blandizzi, C. ; Bernardini, M. C. ; [et al.]

Springer

Cellular and molecular life sciences 45 (1989), S. 734-736

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ISSN: 1420-9071

Keywords: Gastrin release ; rat isolated stomach ; baclofen ; GABA-A receptors

Source: Springer Online Journal Archives 1860-2000

Topics: Biology , Medicine

Notes: Summary In order to investigate the role of peripheral GABA-B receptors, the effects of the putative GABA-B agonist baclofen on immunoreactive gastrin release from an isolated vascularly perfused rat stomach preparation were examined. The vascular infusion of baclofen at graded concentrations induced a dose-dependent increase in gastrin release; this was unaffected by the GABA-B antagonist delta-aminovaleric acid, but was fully prevented by the selective GABA-A antagonist bicuculline as well as by atropine or tetrodotoxin. These results suggest that the stimulant effects of baclofen are mediated by nervous cholinergic structures, associated with GABA-A receptors, and indicate that this GABA-B agonist must be regarded as a partial agonist of peripheral GABA-A receptors.