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  • 1985-1989  (2)
  • 1
    Electronic Resource
    Electronic Resource
    Effects of β-hydroxybutyrate on rat embryos grown in culture (1985)
    Springer
    Cellular and molecular life sciences 41 (1985), S. 273-275 
    Details
    ISSN: 1420-9071
    Keywords: Rat ; embryo ; embryo ; rat ; β-hydroxybutyrate ; diabetes ; maternal ; fetal malformation ; malformation ; fetal ; ketone bodies
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary In order to investigate further the relationship between maternal diabetes and fetal malformation, rat embryos were grown in vitro in the presence of β-hydroxybutyrate, one of the ketone bodies produced by diabetics. At 10 mM, β-hydroxybutyrate produced minor abnormalities and at 20 mM it produced major abnormalities in rat embryos.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02002633
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Identification of two complementation groups in Fanconi anemia (1985)
    Springer
    Somatic cell and molecular genetics 11 (1985), S. 35-41 
    Details
    ISSN: 1572-9931
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Considerable variation can be observed in the clinical presentation of Fanconi anemia (FA) patients and in the degree of sensitivity of their cells to DNA damaging agents. We have examined the hypothesis that genetic heterogeneity underlies this variation by testing for complementation in somatic cell hybrids constructed from FA cells. Hybrids were formed by fusing lymphoblastoid cell lines derived from four different FA patients. Complementation of the cellular defects in FA was tested by examining sensitivity to growth inhibition by mitomycin C(MMC), spontaneous chromosome breakage, and MMC-induced chromosome breakage in the hybrid cells. These studies revealed the presence of at least two complementation groups, suggesting that there may be two or more different FA genes.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01534732
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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