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  • 1980-1984  (2)
  • 1905-1909
  • Adenosine  (1)
  • Brain tumour  (1)
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  • 1980-1984  (2)
  • 1905-1909
Year
Keywords
  • 1
    Electronic Resource
    Electronic Resource
    Adenosine induced fall in glomerular capillary pressure (1981)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 317 (1981), S. 86-89 
    Details
    ISSN: 1432-1912
    Keywords: Glomerular capillary pressure ; Munich-Wistar rat ; Adenosine ; Ureteral obstruction ; Aortic constriction
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The effect of acute ureteral obstruction (UO) and reduction of renal artery pressure (AC) on the adenosineinduced renal vasoconstriction was studied in the Munich-Wistar rat. Infusion of adenosine, 0.05 μmol/min · kg body weight, into the thoracic aorta, was associated with a fall of directly measured glomerular capillary pressure (P gc) from 45.2+1.8 to 32.5+1.7 mm Hg, P〈0.001. Elevation of ureter pressure to 39+2 mm Hg abolished the fall of P gc following adenosine infusion, 51.3+1.7 vs. 50.0+1.3 mm Hg, NS. Reduction of renal artery pressure to 70 mm Hg by an aortic clamp above the renal arteries also prevented the fall of P gc due to adenosine, 36.8+0.9 vs. 36.4+1.8 mm Hg, NS. Administration of indometacin (10 mg/kg i.v.) restored the ability of adenosine to reduce P gc in UO from 41.5+1.1 to 25.9+2.6 mm Hg (P〈0.001) and in AC from 34.0+3.4 to 28.2+75.7 mm Hg (P〈0.02). Since previous studies have demonstrated that in UO and AC renal prostaglandin synthesis is enhanced the effects of indometacin suggest that prostaglandins could be antagonistic to the action of adenosine on the kidney. The data show that the renal vasculature becomes insensitive to the vasoconstrictive action of adenosine during elevated ureter pressure and reduced renal artery pressure.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00506263
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Aphasia and non-verbal intelligence in brain tumour patients (1982)
    Springer
    Journal of neurology 227 (1982), S. 209-218 
    Details
    ISSN: 1432-1459
    Keywords: Aphasia ; Intelligence ; Brain tumour
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung 43 Kranke mit Hirntumoren unterschiedlicher Lokalisation und Art wurden mit dem Aachener Aphasie Test untersucht. Bei den meisten Personen wurde zur gleichen Zeit der Handlungsintelligenzquotient mit Hilfe des Hamburg-Wechsler-Intelligenz-Testes ermittelt. Die Untersuchung hat folgendes ergeben: 1. Sprachstörungen bei Hirntumorkranken sind in der Regel amnestische Aphasien, unabhängig vom prä- oder postsylvischen Sitz der Geschwülste. 2. Personen mit Aphasie haben keine größere Intelligenzstörung als nicht aphasische mit gleicher Hirnschädigung. 3. Die Schwere der Aphasie korreliert nicht mit dem Handlungsintelligenzquotienten. 4. Bei rechtshirnigen Geschwulstträgern korrelieren die Leistungen in dem Untertest Sprachverständnis des Aachener Aphasie Tests mit dem Handlungs-IQ. Dies läßt vermuten, daß dieser Untertest auch visuo-cognitive Fähigkeiten prüft.
    Notes: Summary Patients with primary brain tumours were investigated using the Aachen Aphasia Test and subtests of the Wechsler Adult Intelligence Scale (WAIS). The statistical evaluation of the data from 43 patients indicated that: (1) Aphasia was an amnestic aphasia, independent of the localization of the tumour within the dominant hemisphere; (2) aphasics have no more intellectual impairment than non-aphasics with similar brain lesions; (3) the severity of the aphasic syndrome does not correlate with the WAIS; (4) with right-sided tumours, there was correlation between the speech reception subtest and the WAIS. We conjecture that this subtest tested visuocognitive abilities in patients with right-sided brain lesions.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00313388
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    Paper (German National Licenses)
    Fulltext
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