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  • 1980-1984  (2)
  • Distribution  (1)
  • Pancreatic duct ligation  (1)
  • 1
    Electronic Resource
    Electronic Resource
    Pancreatic duct ligation and pancreatic polypeptide (PP) secretion (1982)
    Springer
    Journal of molecular medicine 60 (1982), S. 211-212 
    Details
    ISSN: 1432-1440
    Keywords: PP secretion ; Pancreatic duct ligation ; PP-Sekretion ; Pankreasgangligatur
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung Die basale und durch eine Testmahlzeit stimulierte Sekretion des Pankreatischen Polypeptids (PP) wurde bei 5 Schweinen 4–6 Wochen nach einer Pankreasgangligatur untersucht, bei 2 Schweinen zusätzlich nach 14 bzw. 16 Wochen. Bei allen Versuchstieren zeigte die Autopsie die erwartete extreme Atrophie des exokrinen Pankreasgewebes. Die basale und postprandiale PP-Sekretion wurde dagegen durch die Pankreasgangligatur nicht beeinflußt.
    Notes: Summary The basal and meal-stimulated secretion of pancreatic polypeptide (PP) were measured before and 4–6 weeks after pancreatic duct ligation in five piglets, in two piglets also after 14 and 16 weeks. In all animals histology revealed the expected extreme atrophy of the exocrine pancreatic tissue. Basal and postprandial PP secretion, however, were not altered after duct ligation.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01715589
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  • 2
    Electronic Resource
    Electronic Resource
    Pancreatic PP cell distribution and hyperplasia (1980)
    Springer
    Virchows Archiv 387 (1980), S. 319-331 
    Details
    ISSN: 1432-2307
    Keywords: Endocrine pancreas ; PP cells ; Immunocytochemistry ; Distribution ; Normal pancreas ; Chronic pancreatitis ; Pancreatic carcinoma
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The endocrine pancreatic tissue from 13 patients with severe chronic pancreatitis, 5 patients with pancreatic duct carcinoma and 4 non-diseased pancreases was analysed by immunocytochemistry and morphometry. The controls revealed two distinct islet types with different regional distribution. The lower dorsal part of the pancreatic head contained islets with irregular outlines and a high number of PP cells (PP-cells 60.4±4.1%; B-cells 29.4±4.6%; A-cells 7.4±1.5%; D-cells 2.8±0.6%). The other parts of the pancreas contained compact islets with only a few PP cells (PP-cells 1.0±0.4%; B-cells 69.3±3.0%; A-cells 24.1±2.1%; D-cells 5.8±0.5%). In chronic pancreatitis the sclerotic tissue of the body and the tail region contained compact islets with altered cell inter-relationships when compared with controls. While the number of B-cells was diminished (48.5%), A and PP cells appeared to be increased in number (42.7 and 4.1%, respectively). Furthermore, ductulo-insular proliferations were conspicuous (nesidioblastosis) with budding-off of small endocrine cell clusters made up predominantly of A and PP cells. In 3 patients with pancreatic carcinoma increased numbers of PP cells and of A cells were found along the advancing edge of the carcinoma. The data emphasize the necessity of taking into consideration regional PP cell distribution in each case in which an increase of PP cells is observed. True hyperplasia is found in chronic pancreatitis and, focally, in some cases with pancreatic carcinoma.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00454835
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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