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  • 1975-1979  (2)
  • 1955-1959
  • Acetylcholinesterase  (1)
  • Bone marrow transplantation  (1)
  • 1
    Electronic Resource
    Electronic Resource
    The use of lymphocyte phosphoglucomutase as a genetic marker in bone marrow transplant recipients (1979)
    Springer
    Annals of hematology 38 (1979), S. 135-137 
    Details
    ISSN: 1432-0584
    Keywords: Knochenmarktransplantation ; Genetischer Marker ; Lymphozyten-Phosphoglukomutase ; Bone marrow transplantation ; Genetic marker ; Lymphocyte phosphoglucomutase
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Summary Lymphocyte phosphoglucomutase can be used as a genetic marker to document successful engraftment in bone marrow transplant recipients. Two patients who underwent marrow transplantation as a treatment for acute leukemia showed a change into donor-type isozyme pattern.
    Notes: Zusammenfassung Phosphoglukomutase aus Lymphozyten kann als genetischer Marker in der Knochenmarktransplantation verwendet werden. Nach Knochenmarktransplantation von zwei Patienten mit akuter Leukose konnte der Übergang in das Iso-Enzymmuster des Spenders beobachtet werden.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01007954
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Effects of dichlorvos (DDVP) inhalation on the activity of acetylcholinesterase in the bronchial tissue of rats (1978)
    Springer
    Archives of toxicology 42 (1978), S. 191-198 
    Details
    ISSN: 1432-0738
    Keywords: Dichlorvos ; Acetylcholinesterase ; Bronchial tissue
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The experiments presented here deal with the effects of the inhalation of dichlorvos [dimethyl-(2,2 dichlorvinyl)-phosphate, DDVP] vapor on acetyl-cholinesterase (ACHE) activity in rat bronchial tissue. Exposure to DDVP concentrations of 0.8 and 1.8 μg/l for 3 days reduced ACHE activity in the bronchial tissue (62.8±0.8 and 51.6±1.6% of the control), but did not elicit any changes in blood ACHE activity (101±4.5% of the control each). Higher concentrations (4.3 μg/l) induced a decline in ACHE activity also in the blood (38.2±1.1% of the control). In the histochemical preparations used to demonstrate CHE activity in bronchial tissue (thiolacetic acid method), a staining of the bronchial glands and smooth muscles characteristic of the enzyme activity was strongly reduced after exposure of the animals to even the lowest dose applied (0.2 μg/l). The question of whether localized inhibition of ACHE in the bronchial tissue might cause increases in airway resistance due to activation of a broncho-bronchial reflex is discussed. This efferent cholinergic mechanism has been found to be at least partly responsible for maintenance of bronchospasm and hypersecretion in chronic obstructive deseases of the respiratory system.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00353711
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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