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  • 1
    Electronic Resource
    Electronic Resource
    A smooth compactification of the space of transfer functions with fixed McMillan degree (1994)
    Springer
    Acta applicandae mathematicae 34 (1994), S. 329-352 
    Details
    ISSN: 1572-9036
    Keywords: 93B25 ; 93C35 ; 32J05 ; 14M15 ; Transfer functions ; compactification ; Quot scheme ; Hermann-Martin curve
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mathematics
    Notes: Abstract It is a classical result of Clark that the space of all proper or strictly properp ×m transfer functions of a fixed McMillan degreed has, in a natural way, the structure of a noncompact, smooth manifold. There is a natural embedding of this space into the set of allp × (m+p) autoregressive systems of degree at mostd. Extending the topology in a natural way we will show that this enlarged topological space is compact. Finally we describe a homogenization process which produces a smooth compactification.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00998684
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Acute effects of clonidine on central and peripheral haemodynamics and plasma renin activity (1972)
    Springer
    European journal of clinical pharmacology 4 (1972), S. 107-114 
    Details
    ISSN: 1432-1041
    Keywords: Clonidine ; circulation ; renin ; symphatetic nerves ; haemodynamics
    Source: Springer Online Journal Archives 1860-2000
    Topics: Chemistry and Pharmacology , Medicine
    Notes: Summary After the intravenous administration of clonidine normo- and hypertensive patients were studied by measurement of central, peripheral and renal hemodynamics, and plasma renin activity. The patients were examined either at rest, or while supine after tilting to 30° for 10 min. In most patients 300 µg of clonidine lowered both the blood and pulse pressures, heart rate, stroke volume and cardiac index, as well as the central venous pressure. In general the peripheral vascular resistance did not change, although it fell in some hypertensive patients. Renal vascular resistance diminished and renal blood flow and glomerular filtration rate sometimes rose, but more frequently remained unchanged. — The results suggest a centrally mediated action of clonidine, possibly on the sympathetic system as they can be interpreted as evidence of inhibition of sympathetic responses. The failure of the peripheral renin level to rise supports this hypothesis.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00562506
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Einfluß von Saralasin auf Plasma Renin Aktivität und Arginin-Vasopressin bei Patienten mit angiotensinabhängiger Hypertonie (1978)
    Springer
    Journal of molecular medicine 56 (1978), S. 625-627 
    Details
    ISSN: 1432-1440
    Keywords: Hypertension ; Plasma renin activity ; Argininevasopressin ; Saralasin ; Hypertonie ; Plasma Renin Aktivität ; Arginin-Vasopressin ; Saralasin
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung Eine zentral stimulierende Wirkung von Angiotensin II (AII) auf die Sekretion von Arginin-Vasopressin (AVP) ist beschrieben worden. Als kompetitiver Blocker von AII fand das Analogon Saralasin (SAR) zum Nachweis einer angiotensinabhängigen Hypertonie Verwendung. Zusätzlich wurde eine partiell agonistische Wirkung von SAR nachgewiesen. Ziel der vorliegenden Studie war es festzustellen, ob die durch SAR induzierte Reninstimulation zu meßbaren AVP-Veränderungen führt. Bei 9 essentiellen Hypertonikern, deren arterieller Blutdruck während einer SAR-Infusion (10 µg/kg/min über 30 min Dauer) signifikant abfiel, wurden vor Beginn und am Ende der Applikation Plasma Renin Aktivität (PRA) und AVP radioimmunologisch bestimmt. Bei allen Patienten führte das depressorisch wirkende SAR zu einem signifikanten Anstieg der PRA, bei 6 Patienten zu einer signifikanten Erhöhung von AVP im Serum. Die unter SAR festgestellten AVP-Anstiege erklären sich möglicherweise durch die periphere depressorische Wirkung, durch eine mangelnde Konzentration am zentralen Rezeptor oder durch eine partielle AII-agonistische zentrale Wirkung.
    Notes: Summary A central stimulatory effect of angiotensin II (AII) on the secretion of arginine-vasopressin (AVP) has been described. The competitive blocker of AII, saralasin (SAR) has been used for diagnostic purposes in angiotensin-dependent hypertension. In addition SAR has a partially agonistic effect. The aim of the present study was to demonstrate whether AVP-levels can be influenced during SAR-induced renin stimulation. In 9 patients with essential hypertension blood pressure dropped significantly under SAR (10 µg/kg/min over a 30 min period). Before and after SAR plasma renin activity (PRA) and AVP were measured by RIA. SAR evoked significant increments of PRA in all patients and of AVP in 6 patients. The increased serum concentrations of AVP following SAR may be explained either by the depressor effect of SAR, its diminished concentration at the central receptor, or a partial AII-agonistic effect.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01477012
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    Paper (German National Licenses)
    Fulltext
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