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  • 1
    Electronic Resource
    Electronic Resource
    On the mechanism of an increase of muscle performance and of vasodilation during emotional stress in man (1978)
    Springer
    Pflügers Archiv 373 (1978), S. 211-218 
    Details
    ISSN: 1432-2013
    Keywords: Mental arithmetic ; Skeletal muscle blood flow ; Evoked contraction of skeletal muscle ; Muscle metabolism ; McArdle's syndrome
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The mechanism of the increase of muscle performance and of vasodilation during emotional stress was studied. The “emotional” increment of voluntary performance does not depend on the level of blood supply to the working muscles, and the effect is maintained under conditions of cessation of arterial inflow. Augmentation of muscle performance is also observed during emotional stress when isometric contraction is evoked by electrical tetanic stimulation of the nerve, when the number of muscle fibres participating in the evoked response does not increase during emotional stress. The “emotional” vasodilation is greatly reduced in patients suffering from McArdle's syndrome, in whom the normal course of glycolysis in muscles is disrupted. It is suggested that acetylcholine liberated from sympathetic fibres causes the activation of glycolysis in muscles, which in its turn induces vasodilation in resting skeletal muscles and increase of muscle performance during emotional stress.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00580826
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Role of ATP as a sympathetic nervous system transmitter in the smoothing of rapid arterial pressure changes (1995)
    Springer
    Bulletin of experimental biology and medicine 120 (1995), S. 1083-1086 
    Details
    ISSN: 1573-8221
    Keywords: ATP ; awake rats ; arterial pressure lability ; sympathetic nervous system
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Arterial pressure lability and its variations were examined in unrestrained rats following selective elimination of adrenergic or purinergic sympathetic influences on the circulatory system. Both the α1-andrenoceptor blocker prazosin and the nonselective α-adrenoceptor blocker phentolamine lowered the arterial pressure without affecting its lability. When P2x purine receptors were desensitized with α,β-methyleneATP, the resulting pronounced hypotension was accompanied by a two-fold increase in the lability of mean arterial pressure.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02445470
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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