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  • Cultured smooth muscle cells  (3)
  • Guinea-pig taenia coli  (2)
  • ATP release  (1)
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  • 1
    Electronic Resource
    Electronic Resource
    ATP release by angiotensin II from segments and cultured smooth muscle cells of guinea-pig taenia coli (1996)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 354 (1996), S. 796-799 
    Details
    ISSN: 1432-1912
    Keywords: Key words ATP release ; Angiotensin II ; Losartan ; AT1-receptor ; Cultured smooth muscle cells ; Guinea-pig taenia coli
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Effects of angiotensin II on ATP release were evaluated in segments and cultured smooth muscle cells from the guinea-pig taenia coli. In the segments, angiotensin II (0.3–3 μM) elicited release of ATP which was blocked by losartan and SC-52458, non-peptide angiotensin II type-1 receptor (AT1)-antagonists, but not by PD-123319, an AT2-antagonist. In superfused cultured cells, 10 μM angiotensin II likewise elicited release of ATP. Again the response was blocked by losartan and SC-52458 but not by PD-123319. These findings suggest that angiotensin II releases ATP from the smooth muscles by activation of angiotensin II-, presumably AT1-, receptors.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00166908
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    ATP release caused by bradykinin, substance P and histamine from intact and cultured smooth muscles of guinea-pig vas deferens (1998)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 357 (1998), S. 240-244 
    Details
    ISSN: 1432-1912
    Keywords: Key words Non-neuronal ATP release ; Bradykinin ; Substance P ; Histamine ; Guinea-pig vas deferens ; Cultured smooth muscle cells ; Autacoid.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Histamine (60 μM) produced ATP release from segments of guinea-pig vas deferens which was blocked by pyrilamine and triprolidine, H1-blockers, but not by ranitidine, an H2-blocker. The evoked-release was inhibited by the mitochondrial inhibitors, carbonyl cyanide-m-chlorophenylhydrazone (CCCP) and oligomycin. Bradykinin (BK) and substance P (SP) also caused substantial and moderate release of ATP, respectively. The BK-evoked release of ATP was inhibited by HOE140, a B2-antagonist, but not by [Des-Arg10] HOE140, a B1-antagonist. On the other hand, VIP, angiotensin II (AII) and cholecystokinin-octapeptide (CCK-8) failed to elicit a measurable release of ATP. Histamine and BK also enhanced the release of ATP from superfused cultured smooth muscle cells. These results suggest that ATP may be released as an autacoid from the smooth muscles in the presence of these chemical mediators.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/PL00005163
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    ATP release by angiotensin II from segments and cultured smooth muscle cells of guinea-pig taenia coil (1996)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 354 (1996), S. 796-799 
    Details
    ISSN: 1432-1912
    Keywords: ATP release ; Angiotensin II ; Losartan ; AT1-receptor ; Cultured smooth muscle cells ; Guinea-pig taenia coli
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Effects of angiotensin II on ATP release were evaluated in segments and cultured smooth muscle cells from the guinea-pig taenia coli. In the segments, angiotensin II (0.3–3 μM) elicited release of ATP which was blocked by losartan and SC-52458, non-peptide angiotensin II type-1 receptor (AT1)-antagonists, but not by PD-123319, an AT2-antagonist. In superfused cultured cells, 10μM angiotensin II likewise elicited release of ATP Again the response was blocked by losartan and SC-52458 but not by PD-123319. These findings suggest that angiotensin II releases ATP from the smooth muscles by activation of angiotensin II-, presumably ATE1-, receptors.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00166908
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
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