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  • 1
    Electronic Resource
    Electronic Resource
    Construction of a brewer's yeast having α-acetolactate decarboxylase gene from Acetobacter aceti ssp. xylinum integrated in the genome
    Amsterdam : Elsevier
    Journal of Biotechnology 32 (1994), S. 173-178 
    Details
    ISSN: 0168-1656
    Keywords: A. aceti ssp. xylinum ; Brewer's yeast ; Glycolytic promoter ; Integration ; α-Acetolactate decarboxylase
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Process Engineering, Biotechnology, Nutrition Technology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1016/0168-1656(94)90180-5
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    High level expression of α-acetolactate decarboxylase gene from Acetobacter aceti subsp. xylinum in brewing
    Amsterdam : Elsevier
    Journal of Biotechnology 37 (1994), S. 45-48 
    Details
    ISSN: 0168-1656
    Keywords: Acetobacter aceti subsp. xylinum ; Brewer's yeast ; Diacetyl ; Glycolytic promoter ; α-Acetolactate decarboxylase
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Process Engineering, Biotechnology, Nutrition Technology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1016/0168-1656(94)90201-1
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Cloning and expression of the gene encoding α-acetolactate decarboxylase from Acetobacter aceti ssp. xylinum in brewer's yeast
    Amsterdam : Elsevier
    Journal of Biotechnology 32 (1994), S. 165-171 
    Details
    ISSN: 0168-1656
    Keywords: Acetobacter aceti ssp. xylinum ; Brewer's yeast ; Cloning ; α-Acetolactate decarboxylase
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Process Engineering, Biotechnology, Nutrition Technology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1016/0168-1656(94)90179-1
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    Paper (German National Licenses)
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  • 4
    Electronic Resource
    Electronic Resource
    Involvement of the median preoptic nucleus in the regulation of paraventricular vasopressin neurons by the subfornical organ in the rat (1989)
    Springer
    Experimental brain research 76 (1989), S. 47-54 
    Details
    ISSN: 1432-1106
    Keywords: Subfornical organ ; Median preoptic nucleus ; Paraventricular nucleus ; Vasopressin neuron ; Angiotensin II ; Saralasin
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Extracellular recordings in urethane-anesthetized male rats indicated that electrical stimulation of the subfornical organ (SFO) alters the activity of 54 out of 62 phasically firing neurosecretory neurons in the hypothalamic paraventricular nucleus (PVN); 44 cells demonstrate an increase in excitability; 10 cells display a depression in their activity. In 14 out of 38 PVN cells tested, SFO stimulation-evoked excitations were abolished by pretreatment with the angiotensin II (ANG II) antagonist, saralasin (Sar), in the region of the median preoptic nucleus (MnPO). Inhibitory responses (n=7) were not affected. Microinjection of ANG II into the region of the SFO produced either a facilitation (n=28) or no effect (n=6) on the excitability of phasically active PVN neurosecretory cells and the facilitatory effect of 9 out of 23 cells tested was prevented by pretreatment with Sar in the region of the MnPO. All the PVN cells which had excitatory responses to either electrical (n=7) or chemical (n=9) stimulation of the SFO that were blocked following the pretreatment could also be activated by intravenous administration of ANG II. Furthermore, this activation was blocked (n=10) or attenuated (n=6) by pretreatment with Sar in the region of the MnPO. These results show an involvement of both the MnPO and the SFO for the regulation of excitability of putative vasopressin (VP)-secreting PVN neurons, and suggest that MnPO neurons sensitive to ANG II may relay activation of SFO neurons by circulating ANG II to putative VP-secreting PVN neurons which result in enhanced excitability.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00253622
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    Paper (German National Licenses)
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  • 5
    Electronic Resource
    Electronic Resource
    Efferent pathways from the region of the subfornical organ to hypothalamic paraventricular nucleus: an electrophysiological study in the rat (1986)
    Springer
    Experimental brain research 62 (1986), S. 509-514 
    Details
    ISSN: 1432-1106
    Keywords: Subfornical organ ; Paraventricular nucleus ; Angiotensin II ; Acetylcholine ; Vasopressin neuron
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Twenty-three neurons in the region of the subfornical organ (SFO) were antidromically activated by electrical stimulation of the hypothalamic paraventricular nucleus (PVN) in male rats under urethane anesthesia. Microiontophoretically (MIPh) applied angiotensin II (AII) excited the activity of all units in the region of the SFO and the effect of AII was blocked by MIPh applied saralasin (Sar), an AII antagonist, but not by atropine (Atr), a muscarinic antagonist. In these units, 12 were also excited by MIPh applied acetylcholine (ACh) while 11 were not affected and the effect of ACh was attenuated by not only MIPh applied Atr, but also Sar, suggesting that not only neurons specific for AII, but also neurons sensitive to both AII and ACh project to the PVN in the region of the SFO. Intravenously administered AII excited the activity of both types of units in the region of the SFO. Microinjected AII or ACh into the region of the SFO excited the activity of putative vasopressin (VP)-secreting units in the PVN. These results suggest that neurons projecting to the PVN in the region of the SFO may act to enhance the activity of putative VP-secreting neurons in the PVN in response to circulating AII.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00236029
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    Paper (German National Licenses)
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  • 6
    Electronic Resource
    Electronic Resource
    Subfornical organ and hypothalamic paraventricular nucleus connections with median preoptic nucleus neurons: an electrophysiological study in the rat (1987)
    Springer
    Experimental brain research 68 (1987), S. 579-585 
    Details
    ISSN: 1432-1106
    Keywords: Subfornical organ ; Median preoptic nucleus ; Paraventricular nucleus ; Angiotensin II ; Vasopressin neuron
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The role of pathways from the subfornical organ (SFO) to the hypothalamic paraventricular nucleus (PVN) through the median preoptic nucleus (MnPO) in regulating the activity of putative vasopressin (VP)-secreting neurons in the PVN was examined in urethane-anesthetized male rats. The activity of the majority (79%) of SFO neurons antidromically identified as projecting to the MnPO was excited by microiontophoretically (MIPh) applied angiotensin II (ANG II) and the effect was blocked by MIPh-applied saralasin (Sar), an ANG II antagonist. Identified SFO neurons that were excited by MIPh-applied ANG II were also excited by intravenously administered ANG II. Electrical stimulation of the SFO produced orthodromic excitation (48%) or inhibition (24%) of the activity of MnPO neurons antidromically identified as projecting to the PVN. Identified MnPO neurons that were excited by SFO stimulation were also excited by MIPh-applied ANG II, while the remaining neurons were not affected. The excitatory responses to SFO stimulation and to MIPh-applied ANG II were both blocked by MIPh-applied Sar, whereas the inhibitory responses to SFO stimulation were not affected. ANG II injected into the region of the SFO produced either an excitation (55%) or no effect (45%) on the activity of identified MnPO neurons. Electrical stimulation of the MnPO produced orthodromic excitation (27%) or inhibition (23%) of the activity of putative VP-secreting PVN neurons. ANG II injected into the region of the MnPO produced either an excitation (31%) or no effect (69%) on the activity of putative VP-secreting PVN neurons. These observations reveal some possible interconnections between three brain regions and suggest that circulating ANG II excites a population of neurons projecting from the SFO to the MnPO, and that these neurons themselves release ANG II as an excitatory transmitter on part of MnPO neurons projecting to the PVN, thereby causing enhanced activity of putative VP-secreting PVN neurons.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00249800
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    Paper (German National Licenses)
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