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  • Arteriovenous malformation  (1)
  • Cerebellar cortical atrophy  (1)
  • Concanavalin A  (1)
  • 1
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    Biochimica et Biophysica Acta (BBA)/Biomembranes 599 (1980), S. 301-314 
    ISSN: 0005-2736
    Keywords: (Hepatoma) ; Agglutination ; Cell surface ; Concanavalin A ; Proteoglycan
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology , Medicine , Physics
    Type of Medium: Electronic Resource
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  • 2
    ISSN: 1432-1920
    Keywords: Key words Spinocerebellar ataxia type 6 ; Cerebellar cortical atrophy ; Magnetic resonance imaging
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract We describe the MRI findings in three Japanese patients with spinocerebellar ataxia type 6 (SCA6) in which a polymorphic CAG repeat was identified in the gene encoding the α1A voltage-dependent P/Q-type Ca2+ channel subunit (CACNL1A4). All showed slowly progressive cerebellar ataxia and mild pyramidal signs. Neuroradiologically, they had moderate cerebellar atrophy, most prominently in the superior vermis, whereas the brain stem appeared to be spared. No abnormal signal intensity was identified.
    Type of Medium: Electronic Resource
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  • 3
    ISSN: 0942-0940
    Keywords: Arteriovenous malformation ; computer tomography ; intracerebral haemorrhage
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary We studied the CT findings of 23 AVM cases confirmed by cerebral angiography and their clinical findings, and discussed mainly the lesions of ruptured AVM in the acute stage. The 10 cases, in which CT was performed within 7 days after the onset of a ruptured AVM, presented with blood stained CSF except for two cases. All of the 10 cases showed intracerebral haemorrhages in the CT findings, and four of these cases were associated with ventricular rupture. In the plain CT of these 10 cases, high density in the cerebral cistern, such as seen in the acute stage of ruptured intracranial aneurysms, was not found except for 1 case. In the enhanced CT within 7 days after the onset of ruptured AVM, no enhanced findings were found in eight cases. On the contrary, in 12 cases in which CT was performed later than the 8th day after the onset, enhanced findings were found in 11 cases. It has been previously reported that SAH is seen in the lesion of ruptured AVM in the acute stage. From our CT findings of ruptured AVM in the acute stage, however, it was concluded that the lesion of a ruptured AVM in the acute stage does nat cause SAH but an intracerebral haematoma or ventricular rupture and that blood may enter the CSF only secondarily.
    Type of Medium: Electronic Resource
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