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  • 1
    Electronic Resource
    Electronic Resource
    Neuronal ceroidosis (Ceroid-lipofuscinosis) in a Blue Heeler dog (1986)
    Springer
    Acta neuropathologica 69 (1986), S. 161-164 
    Details
    ISSN: 1432-0533
    Keywords: Canine neuronal ceroidosis ; Blue Heeler dog ; Autofluorescent intraneuronal granules ; Pleomorphic membranogranular cytosomes ; Central nervous system neurons
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Neuronal ceroidosis was observed in an 18-month-old male Blue Heeler dog which was euthanized after showing a progressive gait and behavior abnormality, depression, paresthesia, and vision deterioration. The brain was slightly atrophic. Histopathology revealed autofluorescent, periodic acid-Schiff, luxol fast blue, and oil red O-positive intracytoplasmic granules in the neurons of the brain and spinal cord. There was a moderate diffuse reduction in the number of cerebellar and cerebrocortical neurons. Ultrastructurally, these neuronal cytosomes were diverse with preponderance of membranous profiles and granular matrix.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00687053
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Experimental spongy degeneration in calves (1977)
    Springer
    Acta neuropathologica 39 (1977), S. 115-127 
    Details
    ISSN: 1432-0533
    Keywords: Spongy degeneration ; Calves ; Experimental hyperammonemia ; Intramyelinic vacuoles
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Experimental reproduction and ultrastructural findings of spongy degeneration of the central nervous system of hyperammonemic calves are described. Hyperammonemia was produced by intravenous infusion of ammonium acetate. Histologic findings were stereotyped in all calves and characterized by widespread vacuolation of white and grey matter of the brain and spinal cord. Electron microscopy revealed widespread intramyelinic vacuoles, some expansion of extracellular spaces and swollen mitochondria. There were minimal changes in neurons, axons and glia, and little evidence of myelin breakdown. Hyperammonemia may be one of the pathogeneses involved in the CNS spongy degeneration in man and domestic animals affected with hepatocerebral diseases and some hereditary disorders.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00703317
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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