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  • Entopenduncular nucleus (Pallidum)  (1)
  • Habituation  (1)
  • Multiple congenital anomalies  (1)
  • 1
    Electronic Resource
    Electronic Resource
    Functional influence of the central noradrenergic system on the skin conductance activity in rats
    Amsterdam : Elsevier
    Schizophrenia Research 13 (1994), S. 145-150 
    Details
    ISSN: 0920-9964
    Keywords: 6-Hydroxydopamine ; Habituation ; Noradrenalin ; Skin conductance response ; Vigilance
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0920-9964(94)90095-7
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    The mode of pallido-thalamic transmission investigated with intracellular recording from cat thalamus (1978)
    Springer
    Experimental brain research 33 (1978), S. 493-507 
    Details
    ISSN: 1432-1106
    Keywords: VL-VA neurons ; Entopenduncular nucleus (Pallidum) ; Monosynaptic inhibition
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Pallido-thalamic transmission was studied by intracellular recording from neurons in the ventrolateral (VL) and ventroanterior (VA) nuclei of the thalamus in cats anesthetized with pentobarbital. Stimulation of the entopeduncular nucleus (ENT) produced short latency, inhibitory postsynaptic potentials in the VL-VA neurons (1.60 ms on average). When stimuli were applied closer to the VL-VA region along the pallido-thalamic pathway, i.e., to the rostral Forel's field, the IPSP latency was significantly reduced. Linear regression analysis of the IPSP latency against conduction distance between different stimulating and recording positions indicated that the IPSP was produced through a monosynaptic pathway at a conduction velocity of 5 to 11 m/s. The neurons which received IPSPs from the ENT distributed in the rostromedial VL and in the rostral VA, whereas relay cells responding only to the contralateral brachium conjunctivum were found in the caudal VL and in the dorsolateral portion of the rostral VL-VA complex. Reciprocal convergence of pallidal and cerebellar impulses were observed in only a small number of cells, which were located in the border between the two neuron groups. Recording of extracellular field potentials and focal stimulation within and around the rostral VL also indicated that the fiber potentials arose from the ENT nucleus and propagated along a bundle of fibers which terminated within the rostromedial VL-VA complex. These results are all explicable by assuming that the entopeduncular neurons are inhibitory in nature and so inhibit thalamic neurons monosynaptically.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00235570
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    A case of cebocephaly-holoprosencephaly with an aberrant adenohypophysis (1987)
    Springer
    Child's nervous system 3 (1987), S. 251-254 
    Details
    ISSN: 1433-0350
    Keywords: Holoprosencephaly ; Cebocephaly ; 7q deletion ; Aberrant adenohypophysis ; Multiple congenital anomalies
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract An autopsy case of cebocephaly-holoprosencephaly at 27 weeks' gestation is reported. Chromosome analysis revealed a 46, XX,-7, +der(7), t(7; 13) (q32; q34) pat karyotype. Pathological examination disclosed a hypoplastic aberrant adenohypohysis, in which immunohistochemical localization of four anterior pituitary hormones (ACTH, GH, PRL, TSH-β) was demonstrated. Malformation of the pituitary gland in holoprosencephaly can include ectopic adenohypophysis as well as pituitary dysgenesis, so a careful search for adenohypophysis should be made in future cases for a better understanding of endocrine dysgenesis associated with holoprosencephaly.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00274062
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    Paper (German National Licenses)
    Fulltext
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