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  • Growth  (1)
  • Key words Hyperglycemia  (1)
  • Nonlinear renewal theory  (1)
  • 1
    Electronic Resource
    Electronic Resource
    A mathematical analysis of small mammal populations (1987)
    Springer
    Journal of mathematical biology 25 (1987), S. 263-274 
    Details
    ISSN: 1432-1416
    Keywords: Population biology ; Nonlinear renewal theory ; Population waves: synchronization ; Rarefaction and compression
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Mathematics
    Notes: Abstract Populations of Microtus montanus, the montane vole, have been extensively studied. It is known that their reproductive activity is closely linked to the availability of the chemicals in growing plants. We use a mathematical model here to study how the length of the vegetative season and the natural reproduction rhythm of voles are involved in the long term dynamics of the population numbers. In particular, we use data obtained from Timpie Springs, Utah, and from Jackson Hole, Wyoming, to formulate a model. The novelty of this model is its use of littering curves that highlight the temporally discrete nature of vole reproduction. The model shows how the timing of the vegetative season can influence vole population sizes.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00276436
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Overexpression of insulin-like growth factor binding protein-1 in transgenic mice (2000)
    Springer
    Pediatric nephrology 14 (2000), S. 567-571 
    Details
    ISSN: 1432-198X
    Keywords: Key words Hyperglycemia ; Glucose intolerance ; Diabetes ; Metabolic effects ; Insulin-like growth factors ; Growth
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract  Overexpression of insulin-like growth factor-1 binding protein (IGFBP-1) in transgenic mice has provided insight into the physiological role of this binding protein in modulating the metabolic and growth-promoting effects of the IGFs. IGFBP-1 transgenic mice demonstrate both intrauterine and postnatal growth retardation. Organ weight was proportionately reduced relative to body weight in most organs, with the exception of the brain, which was disproportionately small in transgenic mice. There were no gross neurological manifestations of the reduction in brain size. Transgenic mice also demonstrated fasting hyperglycemia, impaired glucose tolerance, and modest insulin resistance in skeletal muscle and hepatic tissue. From these data, we can conclude that overexpression of IGFBP-1 results in inhibition of IGF action and in profound impairment of brain development, modest inhibition of fetal and postnatal growth, and inhibition of the metabolic effects of the IGFs. Increased expression of IGFBP-1 has been documented in a variety of situations, such as fetal nutritional deprivation and hypoxia, and has been considered to be a marker of metabolic disturbances that cause fetal growth retardation. The observations in IGFBP-1 transgenic mice suggest expression of IGFBP-1 may itself contribute to the growth retardation and impaired fetal brain development.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s004670000347
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    Paper (German National Licenses)
    Fulltext
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