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  • 1
    Electronic Resource
    Electronic Resource
    OESTROGEN RECEPTORS AND CARDIOVASCULAR DISEASE (1996)
    Oxford, UK : Blackwell Publishing Ltd
    Clinical and experimental pharmacology and physiology 23 (1996), S. 0 
    Details
    ISSN: 1440-1681
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: 1. Animal and human studies indicate that female sex hormones are protective against the development of coronary atherosclerosis.2. Experiments on vascular smooth muscle cells suggest a direct effect of oestrogen on vascular tissue. Although oestrogen receptors (ER) in vascular tissues and an association between ER stimulation and physiological effects have been demonstrated, the cellular mechanisms underlying the effects of oestrogen on the arterial wall remain to be defined.3. Elucidation of the cellular mechanism underlying the effects of oestrogen on vascular smooth muscle will provide insights into coronary atherosclerosis and will provide an opportunity to apply the protective effects of oestrogen to women in older age groups and to the male population.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1440-1681.1996.tb02775.x
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Properties of the Na+-Ca2+ Antiport of Heart Mitochondriaa (1996)
    Oxford, UK : Blackwell Publishing Ltd
    Annals of the New York Academy of Sciences 779 (1996), S. 0 
    Details
    ISSN: 1749-6632
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Natural Sciences in General
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1749-6632.1996.tb44836.x
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Familial juvenile polyposis coli with congenital heart disease (1990)
    Springer
    Pediatric surgery international 5 (1990), S. 429-432 
    Details
    ISSN: 1437-9813
    Keywords: Polyposis coli ; Juvenile polyp ; Gastrointestinal hereditary polyps ; Congenital heart disease
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Seven cases of familial juvenile polyposis coli (FSPC) and one case of familial adenomatous polyposis coli (FAPC) were described in two generations of a family. Of five siblings, one had FJPC with mitral insufficiency, one had Fallot's tetralogy, and the other three, also with FJPC, had had pulmonic stenosis and died. Congenital heart disease was observed in neither the father nor the aunt. The grandfather also had FAPC, without congenital heart disease. To the best of our knowledge, this association has not previously been reported in the literature. For this paper members of the family were studied and the literature was reviewed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00174342
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    Paper (German National Licenses)
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  • 4
    Electronic Resource
    Electronic Resource
    Cation transport systems in mitochondria: Na+ and K+ uniports and exchangers (1994)
    Springer
    Journal of bioenergetics and biomembranes 26 (1994), S. 519-526 
    Details
    ISSN: 1573-6881
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology , Physics
    Notes: Abstract It is now well established that mitochondria contain three antiporters that transport monovalent cations. A latent, allosterically regulated K+/H+ antiport appears to serve as a cation-extruding device that helps maintain mitochondrial volume homeostasis. An apparently unregulated Na+/H+ antiport keeps matrix [Na+] low and the Na+-gradient equal to the H+-gradient. A Na+/Ca2+ antiport provides a Ca2+-extruding mechanism that permits the mitochondrion to regulate matrix [Ca2+] by balancing Ca2+ efflux against influx on the Ca2+-uniport. All three antiports have well-defined physiological roles and their molecular properties and regulatory features are now being determined. Mitochondria also contain monovalent cation uniports, such as the recently described ATP- and glibenclamide-sensitive K+ channel and ruthenium red-sensitive uniports for Na+ and K+. A physiological role of such uniports has not been established and their properties are just beginning to be defined.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00762736
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    Paper (German National Licenses)
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