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  • 1
    Electronic Resource
    Electronic Resource
    Chichester [u.a.] : Wiley-Blackwell
    Developmental Genetics 3 (1982), S. 235-245 
    ISSN: 0192-253X
    Keywords: Drosophila ; chitin biosynthesis ; lethalcryptocephal mutation ; Life and Medical Sciences ; Genetics
    Source: Wiley InterScience Backfile Collection 1832-2000
    Topics: Biology
    Notes: Estimation of chitin deposition in the pupal and adult cuticles of adult Drosophila melanogaster during the pupal period is described. The timing of the periods of chitin deposition is compared with that deduced by previous workers using electron microscopy. The hypothesis that lethalcryptocephal mutant homozygotes are unable to evert their cephalic complexes at pupation because of excess chitin deposition is examined. The data obtained show no evidence that the mutation has any effect on chitin deposition.
    Additional Material: 4 Ill.
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
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  • 2
    Electronic Resource
    Electronic Resource
    Chichester [u.a.] : Wiley-Blackwell
    Developmental Genetics 5 (1984), S. 103-114 
    ISSN: 0192-253X
    Keywords: Drosophila ; pupation ; larval moults ; Lethalcryptocephal ; Life and Medical Sciences ; Genetics
    Source: Wiley InterScience Backfile Collection 1832-2000
    Topics: Biology
    Notes: The events of normal pupation in Drosophila melanogaster are described in detail from the time of gas bubble expulsion until the completion of pupation with the eversion of the cephalic complex. The importance of the internal gas bubble for posterior movement of the prepupa is examined and its relation to the expulsion of the larval mouthparts and the creation of the anterior gas space described.The phenotype of lethalcryptocephal homozygotes, which characteristically cannot evert their heads, is re-examined. Observations of larval lethality and multiple mouthparts in 1 (2)crc larvae and pupae are described. These new aspects of the mutant phenotype are discussed with respect to the abnormalities of pupation. Fristrom's hypothesis that the basic mutant lesion is an increased stiffness of the pupal cuticle due to an excess chitin deposition is re-evaluated.
    Additional Material: 5 Ill.
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
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