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  • 1
    Electronic Resource
    Electronic Resource
    Defense Mechanisms of the Lungs (1975)
    Palo Alto, Calif. : Annual Reviews
    Annual Review of Physiology 37 (1975), S. 325-350 
    Details
    ISSN: 0066-4278
    Source: Annual Reviews Electronic Back Volume Collection 1932-2001ff
    Topics: Medicine , Biology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1146/annurev.ph.37.030175.001545
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Effect of eicosapentaenoic acid in asthma (1988)
    Oxford, UK : Blackwell Publishing Ltd
    Clinical & experimental allergy 18 (1988), S. 0 
    Details
    ISSN: 1365-2222
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: The role of arachidonic acid metabolites in the pathogenesis of airway inflammation and clinical asthma is currently unknown. The addition of eicosapentaenoic acid (EPA) to the diet of humans has been shown to generate metabolites that are less potent than their arachidonic acid counterparts. The substitution of EPA for arachidonic acid metabolites in patients might cause a decrease in airway inflammation and an improvement in clinical asthma. We studied the effect of addition of EPA to the diet of twelve asthmatic patients. Standard clinical evaluations and pulmonary function tests were done on weeks 0, 3, 6, 10, 12 and 14. Patients ingested either low-dose EPA (0.1 g/day) or high-dose EPA (4.0 g/day) from weeks 6–14 (total of 8 weeks). There was no difference in clinical status or pulmonary function between groups at the start of the study. There was no change in clinical status or pulmonary function between or within groups at the end of 8 weeks of EPA ingestion.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2222.1988.tb02857.x
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Antigen-induzierte Reflexbronchokonstriktion beim Hund (1974)
    Springer
    Lung 150 (1974), S. 277-279 
    Details
    ISSN: 1432-1750
    Keywords: Vagal Reflexbronchoconstriction ; Experimental Canine Asthma
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung Die akute Antigen-induzierte Bronchokonstriktion in einem experimentellen Asthma-Modell bei Hunden kommt im wesentlichen durch eine vago-vagale Reflexbronchokonstriktion zustande. Bei selektiver Blockierung entweder afferenter oder efferenter Vagusbahnen ist die durch spezifische Antigene induzierte Bronchokonstriktion reversibel. Unilaterale Antigen-Exposition führt zu bilateraler Bronchokonstriktion. In bronchographischen Untersuchungen mit Tantal-Puder konnte gezeigt werden, daß das Verteilungsmuster der Bronchokonstriktion nach Antigen-Inhalation derjenigen nach Elektrostimulation des Nervus vagus gleicht.
    Notes: Abstract In experimental asthma in dogs, the major component of the acute bronchoconstrictive response to antigen inhalation appears to involve a vagally mediated reflex. Selective afferent or efferent vagal blockade reversed the bronchoconstriction induced by specific antigens. Unilateral antigen inhalation resulted in bilateral bronchoconstriction. In bronchographic studies using powdered tantalum, we found a similar distribution of bronchoconstriction after antigen inhalation as during electrical stimulation of the vagus nerves.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02179330
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    Paper (German National Licenses)
    Fulltext
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