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Effects of Antemortem Injected Crude Papain in Chicken Muscle (1985)

BROOKS, BARBARA A. ; KLASING, KIRK C. ; REGENSTEIN, JOE M.

Oxford, UK : Blackwell Publishing Ltd

Journal of food science 50 (1985), S. 0

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ISSN: 1750-3841

Source: Blackwell Publishing Journal Backfiles 1879-2005

Topics: Agriculture, Forestry, Horticulture, Fishery, Domestic Science, Nutrition , Process Engineering, Biotechnology, Nutrition Technology

Notes: The effects of antemortem injected crude papain in chicken muscle on muscle protein hydrolysis were investigated. White Leghorn hens were injected intravenously prior to slaughter with crude papain or heat denatured crude papain. Collagen solubility (hydroxyproline) at 60°C was 210% of the heat denatured crude papain control. SDS-PAGE revealed no difference between treatments. Crude papain was unable to penetrate intact cells at physiological temperatures. A small but significant increase in muscle TCA soluble tyrosine was observed due to antemortem papain treatment. Postmortem papain treatment resulted in a 14 fold increase in tyrosine. Little if any intracellular muscle protein hydrolysis occurred as a result of antemortem papain treatment. Collagen hydrolysis may be the major contributor to antemortem papain induced meat tenderness.

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1111/j.1365-2621.1985.tb10479.x

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Cytokine-induced neutrophil chemoattractant production by primary rat alveolar type II cells (1995)

Crippen, Tawni L. ; Klasing, Kirk C. ; Hyde, Dallas M.

Springer

Inflammation 19 (1995), S. 575-586

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ISSN: 1573-2576

Source: Springer Online Journal Archives 1860-2000

Topics: Medicine

Notes: Abstract This study was designed to determine the production of the chemokine cytokine-induced neutrophil chemoattractant (CINC) by primary rat alveolar type II (ATII) cells upon stimulation with exogenous and endogenous proinflammatory factors. Cultures of primary rat ATII cells were exposed to lipopolysaccharide (LPS), interleukin-1 beta (IL-1β) or tumor necrosis factor-alpha (TNFα) over a 16 hour period and the production of CINC both apically and basolaterally was measured by ELISA. Compared to unstimulated (UNS) cultures, LPS, IL-1β and TNFα were found to significantly increase the level of CINC detected in culture by two, four and sixteen hours post stimulation, respectively. ATII cells also demonstrated a polar secretion of CINC. The accumulation of CINC basolaterally was significantly more than apically; 133%, 45%, 117% and 123% for UNS, IL-1β, LPS and TNFα respectively. We demonstrated that primary rat ATII cells may participate in the chemokine network during inflammation by the production of CINC upon stimulation with endogenous and exogenous factors.