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  • 1
    Electronic Resource
    Electronic Resource
    Tolerance induction to rofecoxib in a patient with Bartter's syndrome (2004)
    Oxford, UK : Munksgaard International Publishers
    Allergy 59 (2004), S. 0 
    Details
    ISSN: 1398-9995
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1398-9995.2004.00435.x
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  • 2
    Electronic Resource
    Electronic Resource
    Evidence for a role of somatostatin (S.) in the autonomic control of the heart
    Amsterdam : Elsevier
    Journal of Steroid Biochemistry 20 (1984), S. 1623 
    Details
    ISSN: 0022-4731
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0022-4731(84)90930-0
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  • 3
    Electronic Resource
    Electronic Resource
    Somatostatin infusion in liver cirrhosis: Glucagon control of glucose homeostasis (1980)
    Springer
    Diabetologia 18 (1980), S. 187-191 
    Details
    ISSN: 1432-0428
    Keywords: Cirrhosis ; somatostatin ; glucagon ; plasma glucose ; glucose intolerance ; insulin
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary In order to evaluate the role of glucagon in blood glucose homeostasis in liver cirrhosis, ten normal subjects and ten cirrhotic patients were infused with somatostatin (500 μg/h for 5 h) with and without glucagon (3 mg/kg/h) administration. Somatostatin infusion brought about a fall in plasma glucose both in normal (37%) and cirrhotic (41%) subjects in the first 90 minutes. In normal subjects, this was followed by a rise in plasma glucose (147±2 mg/dl at 5 h), while in cirrhotics no rise in plasma glucose was observed (50±1 mg/dl at 5 h). Plasma insulin and glucagon levels were suppressed in both normal and cirrhotic subjects. Addition of glucagon to the somatostatin infusion caused a two fold rise in plasma glucose level to 183±12 mg/dl at 4 h in normal subjects; a much smaller increase was found in the cirrhotic group (105±3 mg/dl at 4 h). When the infusion was stopped, plasma glucose fell both in normal and cirrhotic subjects (102±14 and 87±2 mg/dl at 6 h respectively). Subsequently, hyperglucagonaemia recurred in the cirrhotic patients (319 ±31 pg/ml). A rebound of plasma insulin was observed in normal subjects (47±8 μU/ml) which did not occur in the cirrhotics (16±2 μU/ml). Thus when both insulin and glucagon were suppressed by somatostatin infusion, euglycaemia occurred in cirrhotic subjects only when glucagon concentration was restored exogenously. We conclude that glucagon is important in glucose homeostasis in patients with liver cirrhosis.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00251914
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  • 4
    Electronic Resource
    Electronic Resource
    Insulin and glucagon concentrations in portal and peripheral veins in patients with hepatic cirrhosis (1979)
    Springer
    Diabetologia 17 (1979), S. 23-28 
    Details
    ISSN: 1432-0428
    Keywords: Insulin ; glucagon ; portal blood ; glucose ; arginine ; liver cirrhosis
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The portal vein was catheterized via the umbilical vein under local anaesthesia in 10 non-diabetic subjects about to undergo exploratory laparotomy and in 8 patients with liver cirrhosis. Immunoreactive insulin (IRI) and glucagon (IRG) were assayed in portal and peripheral blood before and during IV infusion of glucose (0.33 g/kg) or arginine (25 g). Basal peripheral plasma (IRI) levels were raised in cirrhotic patients (19±2 versus 10±1 μU/ml; P〈0.001). Basal portal insulin values, however, did not differ in the two groups. After glucose cirrhotic patients had higher peripheral insulin concentrations, compared to controls, significant at 45 and 60 minutes. In contrast portal insulin levels were higher in controls than in cirrhotics by 1 minute (403±43 versus 158±38 μU/ml; P〈0.001) and remained so for the 60 minutes of study. Similarly, after arginine cirrhotics had significantly higher peripheral insulin concentrations and lower portal concentrations than controls. Peak portal vein insulin levels were delayed in cirrhotics (168±16 μU/ml at 3 min) compared with controls (413±25 μU/ml at 1 min). In the basal state both portal and peripheral glucagon levels were higher in cirrhotics than control subjects. Unlike in controls, IV glucose did not suppress glucagon secretion in cirrhotic patients. Peripheral plasma glucagon concentrations after arginine were also consistently higher in cirrhotics than controls, but unlike insulin portal venous glucagon levels were also raised (1800±360 pg/ml, cirrhotics; 960±87 pg/ml, controls; P〈0.001; 1 min after arginine infusion). We conclude that insulin secretion is decreased in liver cirrhosis and that the peripheral hyperinsulinaemia observed reflects diminished hormone metabolism. The high plasma glucagon levels observed in cirrhotic patients are the result of pancreatic hypersecretion of glucagon.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01222973
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  • 5
    Electronic Resource
    Electronic Resource
    Chronic active hepatitis and Behçet's syndrome (1985)
    Springer
    Clinical rheumatology 4 (1985), S. 93-96 
    Details
    ISSN: 1434-9949
    Keywords: Behçet's Syndrome ; Chronic Active Hepatitis
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary In a patient with Behçet's syndrome the finding of five times the normal level of serum transaminase accompanied by weakness may be correlated to the syndrome itself. We performed needle biopsy of the liver in our patient which showed piecemeal necrosis and portal lympho-monocytic infiltration extending into the lobule. It is interesting that HLA B5 and B27 antigens were present. Simultaneous occurrence of chronic active hepatitis and Behçet's syndrome may imply the same autoimmune pathogenesis, as we suggest in this work.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02032326
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  • 6
    Electronic Resource
    Electronic Resource
    Relapsing polychondritis with severe aortic insufficiency (1985)
    Springer
    Clinical rheumatology 4 (1985), S. 474-480 
    Details
    ISSN: 1434-9949
    Keywords: Relapsing Polychondritis ; Aortic Insufficiency
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Relapsing polychondritis (RP) is a rare disease characterized by auricular, nasal and respiratory tract chondritis, ocular inflammation, inflammatory polyarthritis and cardiovascular abnormalities. Here we describe a patient with a five-year history of mild nasal and auricular chondritis, which suddenly developed into a severe aortic insufficiency with rest dyspnea. The pathogenesis and the management of this rare connective tissue disease are discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02031904
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  • 7
    Electronic Resource
    Electronic Resource
    Effect of second phase precipitates on recovery and recrystallization behaviour of cold-worked Al2024-SiCp composites (1996)
    Springer
    Journal of materials science 31 (1996), S. 1625-1631 
    Details
    ISSN: 1573-4803
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: Abstract The restoration behaviour of SiC particle reinforced 2024-aluminium composite has been studied under conditions where recrystallization-grain growth and precipitation occur concurrently. An emphasis has been given to understanding the recrystallization-precipitation interaction, in the presence of SiCp, during annealing. The composite was fabricated by a casting route followed by hot extrusion. Samples were cold deformed to a reduction in height of either 30 or 42 per cent following a heat treatment. The kinetics of recrystallization has been studied in the temperature range 100–485°C. Recrystallization has been monitored by optical microscopy, coupled with hardness measurements. It has been observed that the composite has a lower recrystallization temperature compared to that of an unreinforced alloy under identical annealing conditions. Following recrystallization, grain growth has been noticed in the composite. Although the degree of cold deformation showed some effect on the recrystallization temperature of the composite, it was found to be a weak controlling factor for average recrystallized grain size. Results showed that, in spite of the hardness reduction due to recrystallization-grain growth, the hardness of a composite can be partially restored by concurrent solid solution hardening (dissolution of precipitates) and also by the generation of dislocations during quenching to a low temperature.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00357874
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