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  • 1
    Electronic Resource
    Electronic Resource
    SODIUM AND NORADRENALINE IN CEREBROSPINAL FLUID AND BLOOD IN SALT-SENSITIVE AND NON-SALT-SENSITIVE ESSENTIAL HYPERTENSION (1992)
    Oxford, UK : Blackwell Publishing Ltd
    Clinical and experimental pharmacology and physiology 19 (1992), S. 0 
    Details
    ISSN: 1440-1681
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: 1. The effects of dietary sodium on blood pressure and levels of sodium, other electrolytes and noradrenaline (NA) in the cerebrospinal fluid (CSF) and blood of 15 patients with essential hypertension were studied. The CSF and blood sampling was carried out after 7 days of a high salt intake (16–18 g/day) and after 7 days of a low salt intake (1–3 g/day).2. Blood pressure and sodium concentrations in CSF and serum were significantly higher in the high salt period than the low salt period (CSF Na+ concentration: 147.7 ± 0.4 mmol/L vs 145.3 ± 0.5 mmol/L; P 〈 0.001). Levels of CSF pressure and potassium or calcium concentrations were not different between the two periods. Plasma NA and plasma renin activity (PRA) were lower and CSF NA levels tended to be lower in the high salt period.3. The levels and the changes in sodium and NA in CSF were not significantly different between the salt-sensitive (n= 8) and the non-salt-sensitive (n= 7) subjects, but the changes in plasma NA and PRA were smaller in the salt-sensitive subjects.4. These results indicate that the sympathetic nervous system is less suppressed in salt-sensitive subjects during high salt intake. This may be due to altered neural responsiveness to sodium loading rather than being greater increases in sodium concentration in the central nervous system.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1440-1681.1992.tb00444.x
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  • 2
    Electronic Resource
    Electronic Resource
    Hypophosphatemia induced by intravenous administration of saccharated iron oxide (1983)
    Springer
    Journal of molecular medicine 61 (1983), S. 99-102 
    Details
    ISSN: 1432-1440
    Keywords: Saccharated iron oxide ; Hypophosphatemia
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Forty milligrams saccharated iron oxide1 was given intravenously, daily, to nine iron-deficient patients with moderate to severe anemia. During the 14–42 days of treatment, levels of serum inorganic phosphorus (Pi) decreased significantly and in a stepwise manner (before vs 1 week,P〈0.005; 1 week vs 2 weeks,P〈0.01). Seven of the nine patients became hypophosphatemic within 2 weeks and the other two within 4 weeks. In parallel with the decline in serum Pi, the phosphate clearance increased and tubular reabsorption of phosphate decreased. Reversion to normal levels was delayed in patients treated for the longer period.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01496662
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Stroke-prone spontaneously hypertensive rats as an experimental model of malignant hypertension (1980)
    Springer
    Acta neuropathologica 51 (1980), S. 179-184 
    Details
    ISSN: 1432-0533
    Keywords: SHRSP rat ; Fibrinoid arteriolar necrosis ; Microinfarct ; Hemorrhage ; Edema
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary A pathological study of the brain of the stroke-prone spontaneously hypertensive (SHRSP) rats revealed development of fibrinoid necrosis of the wall of the intracerebral arterioles. These arterioles were frequently accompanied by occlusion of the lumen, and occasionally intracerebral hemorrhages and mincroinfarcts. The predominant tissue alteration consisted of rarefaction and cyst formation in the white matter, and rarefaction of the neuropil and preserved neurons in the neocortex at the paramedian region of the cerebral hemispheres. Edema fluid was present in and around the lesions. The tissue degeneration can be interpreted to be the sequela of brain edema. Microinfarcts or hemorrhages are only focal lesions, and are assumed to have minor contribution to the brain sweelling. Widespread expansion of the extracellular space is assumed to be responsible for the brain swelling. Overall vascular changes of the brain, kidney, and other organs were consistent with those found in malignant hypertension.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00687384
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  • 4
    Electronic Resource
    Electronic Resource
    Primary familial amyloidosis with vitreous opacities (1978)
    Springer
    Acta neuropathologica 42 (1978), S. 67-70 
    Details
    ISSN: 1432-0533
    Keywords: Primary familial amyloidosis ; Vitreous opacities ; Meningovascular deposition ; Extensive cerebral infarction
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary A 41-year-old Japanese male with a new type of primary familial amyloidosis was reported. The patient developed vitreous opacities, and later, disturbances in the gastrointestinal and nervous systems. At autopsy, amyloid was observed in the vitreous and the retinal vessels. There were extensive cerebral infarcts and heavy meningo-vascular amyloid deposition. Although the postmortem study revealed slight peripheral nerve degeneration in the lower extremities secondary to amyloid deposition, there was no clinical evidence of polyneuropathy.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01273271
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  • 5
    Electronic Resource
    Electronic Resource
    An ultrastructural study of developing cerebral infarction following bilateral carotid artery occlusion in spontaneously hypertensive rats (1977)
    Springer
    Acta neuropathologica 40 (1977), S. 171-177 
    Details
    ISSN: 1432-0533
    Keywords: Spontaneously hypertensive rats ; Bilateral carotid occlusion ; Acute ischemic brain damage ; Electron microscopy
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary An ultrastructural study of cerebral infarcts in spontaneously hypertensive rats 1–5 h after bilateral carotid artery occlusion was performed. The alterations of the neocortex consisted of shrinkage of the neurons surrounded by swollen astrocytic processes. Distension of the rough endoplasmic reticulum of the neuronal cytoplasm appeared early, while changes of the mitochondria were slight. Though there appeared slight to moderate perivascular astrocytic swelling, endothelial swelling was rare and there was no severe narrowing of the capillary lumen. There were no filling defects of colloidal carbon injected to the blood vessels of the ischemic brains. Ischemic neuronal alterations were proved to develop in the absence of severe morphological changes of the microvasculature in the developing cerebral infarcts in the present experimental model.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00688707
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    Paper (German National Licenses)
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