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  • 1
    Electronic Resource
    Electronic Resource
    Unmet orthodontic treatment need in rural Nigerian adolescents (1997)
    Oxford, UK : Blackwell Publishing Ltd
    Community dentistry and oral epidemiology 25 (1997), S. 0 
    Details
    ISSN: 1600-0528
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Abstract A survey of orthodontic treatment need was carried out among randomly selected rural Nigerian adolescents using the index of orthodontic treatment need. Altogether, 704 subjects (381 boys and 323 girls) aged 12–18 years (mean 14.8, SD 1.79) were recruited in the study. The results indicated that 12.6% of the population were in objective need of orthodontic treatment. Whilst there was a discrepancy in the proportions of Nigerian adolescents needing orthodontic treatment on aesthetic and dental health grounds, girls were found to have a more attractive dental appearance and less orthodontic treatment need than boys. However, the differences were not statistically significant (P 〉 0.05). The correlation between the orthodontist's and the subject's rating of dental appearance was found to be low (r=0.35). The study also provided reliable baseline data for planning orthodontic services in Nigeria especially in areas where there are no dental services.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1600-0528.1997.tb00956.x
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Epitope expression and hyperphosphorylation of tau protein in corticobasal degeneration: differentiation from progressive supranuclear palsy (1995)
    Springer
    Acta neuropathologica 90 (1995), S. 37-43 
    Details
    ISSN: 1432-0533
    Keywords: Key words Corticobasal degeneration ; Tau protein ; Phosphorylation ; Astrocytic plaques ; Oligodendroglial ; inclusions
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Corticobasal degeneration (CBD) is a rare, progressive neurological disorder characterized by widespread neuronal and glial accumulation of abnormal tau protein. Using immunohistochemistry we analyzed tau epitope expression and phosphorylation state in CBD and compared them to cytoskeletal changes in Alzheimer's disease (AD) and progressive supranuclear palsy (PSP). Epitopes spanning the entire length of the tau protein were present in CBD inclusions. An antibody against the alternatively spliced exon 3 did not recognize cytoskeletal lesions in CBD, but did in AD and PSP. Tau epitopes from each region of the molecule were present in cytoskeletal inclusions in CBD, including gray matter astrocytic plaques, gray and white matter threads, and oligodendroglial inclusions. As in AD, tau from CBD was highly phosphorylated. Antibodies that recognized phosphorylated tau epitopes reacted with material from CBD in a highly phosphatase-dependent manner. Again, all types of inclusions contained phosphorylated epitopes. We conclude that abnormal tau protein in CBD comprises the entire tau molecule and is highly phosphorylated, but is distinguished from AD and PSP by the paucity of epitopes contained in the alternatively spliced exon 3.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00294457
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    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Epitope expression and hyperphosphorylation of tau protein in corticobasal degeneration: differentiation from progressive supranuclear palsy (1995)
    Springer
    Acta neuropathologica 90 (1995), S. 37-43 
    Details
    ISSN: 1432-0533
    Keywords: Corticobasal degeneration ; Tau protein ; Phosphorylation ; Astrocytic plaques ; Oligodendroglial inclusions
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Corticobasal degeneration (CBD) is a rare, progressive neurological disorder characterized by widespread neuronal and glial accumulation of abnormal tau protein. Using immunohistochemistry we analyzed tau epitope expression and phosphorylation state in CBD and compared them to cytoskeletal changes in Alzheimer's disease (AD) and progressive supranuclear palsy (PSP). Epitopes spanning the entire length of the tau protein were present in CBD inclusions. An antibody against the alternatively spliced exon 3 did not recognize cytoskeletal lesions in CBD, but did in AD and PSP. Tau epitopes from each region of the molecule were present in cytoskeletal inclusions in CBD, including gray matter astrocytic plaques, gray and white matter threads, and oligodendroglial inclusions. As in AD, tau from CBD was highly phosphorylated. Antibodies that recognized phosphorylated tau epitopes reacted with material from CBD in a highly phosphatase-dependent manner. Again, all types of inclusions contained phosphorylated epitopes. We conclude that abnormal tau protein in CBD comprises the entire tau molecule and is highly phosphorylated, but is distinguished from AD and PSP by the paucity of epitopes contained in the alternatively spliced exon 3.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00294457
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    Paper (German National Licenses)
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  • 4
    Electronic Resource
    Electronic Resource
    The role of cognitive impairment in the quality of life after ischaemic stroke (1996)
    Springer
    Journal of neurology 243 (1996), S. 599-604 
    Details
    ISSN: 1432-1459
    Keywords: Cerebrovascular diseases ; Quality of life ; Cognition
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Many patient- and disease-related factors influence the quality of life (QL) after stroke. Few studies address the direct relation between cognition and quality of life. The objective of this study was to investigate the role of cognitive impairment in the QL of patients after stroke. We evaluated several variables including age, degree of paralysis, cognitive deficits and volume of infarcts, in 129 patients (64 men, 65 women; mean age 63.2, SD 14.6 years), who had been hospitalized a mean of 2.3 (range 0.25–4) years previously with ischaemic stroke. Cognitive function was assessed with the CAMCOG and QL with a visual analogue scale (VAS). Ninety-seven patients (75%) of our sample completed all the tests. Univariate analysis showed that substantial infarct volume, aphasia, impaired motor function, disability relating to activities of daily life, disturbed global functional health and impaired cognitive function were significantly associated with poorer QL. Age, sex and education of the patient, co-morbidity and location of the infarcts were not related to QL. Linear regression analysis revealed that disturbed global functional health, larger volume of infarcts and severity of aphasia were significant independent explanatory factors for poorer QL (adjustedR 2 = 22%). In conclusion, we did not find a significant impact of cognitive impairment on the patients' QL. However, this conclusion is restricted to a relatively well group of stroke survivors.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00900948
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    Paper (German National Licenses)
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  • 5
    Electronic Resource
    Electronic Resource
    Feasibility of cognitive screening of patients with ischaemic stroke using the CAMCOG A hospital-based study (1996)
    Springer
    Journal of neurology 243 (1996), S. 405-409 
    Details
    ISSN: 1432-1459
    Keywords: Cerebral infarct ; Cognition ; CAMCOG ; Aphasia ; Vascular dementia
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Brief screening tests would be convenient for the measurement of cognitive impairment in stroke patients. In these patients aphasia can interfere with test procedures. To evaluate the feasibility of examining cognitive functions in stroke patients we examined 129 patients with an ischaemic stroke using the CAMCOG, a standardised neuropsychological screening test, after an interval of at least 3 months. Most patients (88%) were able to complete the CAMCOG. Patients with severe aphasia were significantly more likely to have an abnormal CAMCOG score than patients without aphasia [relative risk (RR) 4.0, 95% confidence interval (CI) 2.8–5.8]. The group of patients with moderate aphasia was not at higher risk of having an abnormal CAMCOG score than patients without aphasia (RR 1.4, 95% CI 0.6–2.8). Looking for other factors that might correlate with the scores, logistic regression analysis revealed age as the only significant factor for the prediction of the CAMCOG score (odds ratio 4.0, 95% CI 1.2–13.2). We concluded that the CAMCOG can conveniently be used for screening cognitive functions in patients with cerebral infarcts, even if there is moderate aphasia.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00869000
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    Paper (German National Licenses)
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