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  • 1
    Electronic Resource
    Electronic Resource
    Immunologic and non-immunologic release of histamine and tryptase from human heart mast cells (1995)
    Springer
    Inflammation research 44 (1995), S. S22 
    Details
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01674377
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Does heparin in clinical use contain histamine? (1992)
    Springer
    Inflammation research 36 (1992), S. C184 
    Details
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Clinical observations have shown histamine-typical cardiopulmonary reactions immediately after application of heparin. Heparin is produced from animal tissues, rich in mast cells which contain both heparin and histamine. Heparin in clinical use might therefore still contain considerable amounts of histamine. After mixing heparin with human blood,in vitro, an increase in plasma histamine concentration was observed. Additionally, histamine, in various concentrations, could be detected in different heparin batches and separations.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01997328
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Nitric oxide and the cardiovascular actions of histamine (1992)
    Springer
    Inflammation research 36 (1992), S. C187 
    Details
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract We had found that histamine (HA)-induced relaxation of cavian pulmonary artery is selectively inhibited by Nω-methyl-l-arginine (NMA) and thus depends on the synthesis ofl-arginine (ARG)-derived EDRF/nitric oxide (NO). We have now assessed whether ARG-derived NO also mediates the coronary-vasodilating effect of HA in the intact heart. Langendorff guinea pig hearts, vasoconstricted by the thromboxane agonist U46619 (86 nM), responded to HA (bolus intra-aortic injections of 1–30 μg) with dose-dependent increases in coronary flow (20–90%), heart rate and contractility. NMA (92 μM) markedly inhibited the HA-induced increase in coronary flow, but not its positive inotropic and chronotropic effects. A similar inhibition was obtained with the NO scavenging compound, Fe2+-myoglobin. ARG (1 mM) reestablished the coronary-dilating effects of HA in the presence of NMA. Thus, ARG-derived NO contributes significantly to HA-induced coronary-vasodilatation. Inasmuch as the ultimate HA effect on the coronaries depends on the balance between constriction and relaxation, mediated by smooth muscle and endothelial H1-receptors, respectively, as well as relaxation due to smooth muscle H2-receptors, dysfunction of the NO system is likely to precipitate HA-induced vasospasm.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01997329
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    Paper (German National Licenses)
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  • 4
    Electronic Resource
    Electronic Resource
    A radial compression test for plastic flow studies (2000)
    Springer
    Experimental mechanics 40 (2000), S. 7-9 
    Details
    ISSN: 1741-2765
    Keywords: Plastic flow ; radial compression ; plasticine ; metal forming
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: Abstract A hydraulic experimental setup and related instrumentation were developed for testing constitutive models of ideal plastic behavior at large deformations using slow, radial compression of short, hollow cylinders at constant height. At this preliminary stage of development, plasticine rings with initial inner diameters of 25.4 mm were reduced to a final diameter of 9 mm or less. Further development is needed to test first lead and then harder metals.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02327541
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    Paper (German National Licenses)
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