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  • 1
    Electronic Resource
    Electronic Resource
    Ash Residues from Petroleum Catalytic Cracking Feed Stocks (1952)
    s.l. : American Chemical Society
    Analytical chemistry 24 (1952), S. 742-746 
    Details
    ISSN: 1520-6882
    Source: ACS Legacy Archives
    Topics: Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/ac60064a033
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  • 2
    Electronic Resource
    Electronic Resource
    Human herpesvirus 6-meningoencephalitis in an HIV patient with progressive multifocal leukoencephalopathy (2000)
    Springer
    Acta neuropathologica 100 (2000), S. 337-341 
    Details
    ISSN: 1432-0533
    Keywords: Key words Meningoencephalitis ; Human herpesvirus 6 ; Lymphocyte ; Progressive multifocal leukoencephalopathy ; HIV
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Human herpesvirus 6 (HHV6) has been reported as a rare cause of meningoencephalitis and leukoencephalitis. We present an HIV-infected patient with ¶lesions of progressive multifocal leukoencephalopathy (PML), but also meningoencephalitis apparently due to HHV6. Immunohistochemistry for HHV6 antigens and in situ polymerase chain reaction for HHV6 genome showed many positive lymphocytes and microglia in the meningeal and cortical lesions. More importantly, dead and dying neurons were conspicuous; some were undergoing neuronophagia and some displayed evidence of HHV6 infection. A pathogenic role for this almost universal, and usually commensal, virus in inflammatory brain lesions and PML is briefly discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s004010000198
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  • 3
    Electronic Resource
    Electronic Resource
    Neuronal intranuclear hyaline inclusion disease in a nine year old (1986)
    Springer
    Acta neuropathologica 70 (1986), S. 327-332 
    Details
    ISSN: 1432-0533
    Keywords: Neurons ; Hyaline ; Inclusions ; Neurofilaments
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Thirteen previous cases have been reported as neuronal intranuclear hyaline inclusion disease. The majority of patients have presented with movement disorders at less than 12 years of age followed by a progressive worsening of symptoms and, frequently, loss of cognitive function. Death has usually occurred by the second or third decade. Three have presented in the fifth through seventh decade with either movement disorders or dementia. These cases have been linked by the presence of eosinophilic neuronal intranuclear inclusions diffusely within the CNS and in peripheral ganglion cells. The patient in this case report also presented with a rapidly progressive movement disorder and at autopsy showed the characteristic intranuclear inclusions. Investigation of these inclusions did not reveal shared epitopes with neurofilaments or other intermediate filaments.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00686092
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  • 4
    Electronic Resource
    Electronic Resource
    Ultrastructural heterogeneity in cerebral amyloid of Alzheimer's disease (1988)
    Springer
    Acta neuropathologica 76 (1988), S. 613-623 
    Details
    ISSN: 1432-0533
    Keywords: Amyloid ; Alzheimer's disease ; Carbohydrate ; Senile Brain ; Vesicle
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Cerebral amyloid deposits from five patients with presenile or senile cerebral disease of the Alzheimer type were stained with uranyl acetate and lead citrate or with periodic acid-thiocarbohydrazidesilver proteinate, and examined with traditional highresolution electron microscopy and with a goniometer tilting stage. In addition to a carbohydrate-rich matrix, we also consistently found local cell-derived vesicles within plaque and dyshoric amyloid. The most likely source for these vesicles appeared to be degenerate neurites. Amyloid fibrils were intimately associated with plasmalemmata, particularly those of degenerate neurites, which supported a neuronal origin for the amyloid fibril of Alzheimer's disease.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00689601
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  • 5
    Electronic Resource
    Electronic Resource
    Concomitant Creutzfeldt-Jakob and Alzheimer diseases (1991)
    Springer
    Acta neuropathologica 83 (1991), S. 95-98 
    Details
    ISSN: 1432-0533
    Keywords: Creutzfeldt-Jakob disease ; Alzheimer's disease ; Amyloid ; Protease-resistant protein (PrP) ; Panencephalopathic
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary We report a 69-year-old female with cerebral and cerebellar symptomatology of 15-month duration. At autopsy, both panencephalopathic Creutzfeldt-Jakob and plaque-predominant Alzheimer diseases were found. Plaque amyloid was exclusively of the β/A4 type, but abundant abnormal protease-resistant protein was identified by Western blot analysis of brain extracts.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00294437
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  • 6
    Electronic Resource
    Electronic Resource
    Wolman's disease: Ultrastructural evidence of lipid accumulation in central and peripheral nervous systems (1979)
    Springer
    Acta neuropathologica 45 (1979), S. 37-42 
    Details
    ISSN: 1432-0533
    Keywords: Wolman's disease ; Lipid storage ; Peripheral nerve-glia
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary We report the first case of Wolman's disease in which the fine structure of either the peripheral or the central nervous system has been examined. We confirm ultrastructurally the presence of lipid within endothelial and pericytic cells. Several cell types previously believed to be uninvolved in this storage process demonstrate lipid inclusions characteristic of Wolman's disease: perineurial, endoneurial and Schwann cells of peripheral nerve, and oligodendrocytes and astrocytes of the central nervous system.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00691802
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  • 7
    Electronic Resource
    Electronic Resource
    Permanganate oxidation of senile cerebral amyloid and its relationship to AA protein (1982)
    Springer
    Acta neuropathologica 58 (1982), S. 275-278 
    Details
    ISSN: 1432-0533
    Keywords: AA protein ; Amyloid ; Oxidation ; Immunoperoxidase ; Senile brain
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Immunoreactivity for AA protein was rarely detected in briefly fixed amyloid of senile plaques and dyshoric angiopathy, but was not observed in Congophilic angiopathy. Plaque and dyshoric amyloid exhibited variable sensitivity to permanganate oxidation; Congophilic angiopathy was resistant to oxidation. In contrast to systemic amyloid composed of AA protein, the rare immunoreactivity of senile cerebral amyloid was lost with prolonged fixation. This study demonstrates that the fibrillar protein of senile cerebral amyloid differs from that of systemic amyloid of AA type.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00688609
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  • 8
    Electronic Resource
    Electronic Resource
    An ultrastructural analysis of human post-infectious (allergic) encephalomyelitis (1994)
    Springer
    Acta neuropathologica 87 (1994), S. 541-544 
    Details
    ISSN: 1432-0533
    Keywords: Allergic Encephalomyelitis ; Astrocytes ; Demyelination ; Electron microscopy ; Multiple sclerosis
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract A 6-year-old boy developed post-infectious encephalomyelitis and underwent a brain biopsy (10 days after the onset of neurologic symptoms). Electron microscopic analysis of brain showed demyelinated axons, thinly myelinated axons, aberrant remyelination, and numerous phagocytes containing myelin debris. Physical stripping of myeling by pseudopodial extensions of macrophages, as reported in experimental allergic encephalomyelitis, was noted. Hypertrophic and hyperplastic astrocytes were prominent among the phagocytic cells and played an unexpectedly active role in demyelination.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00294183
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  • 9
    Electronic Resource
    Electronic Resource
    Neuronal lipidosis and neuroaxonal dystrophy in cerebro-hepato-renal (Zellweger) syndrome (1987)
    Springer
    Acta neuropathologica 73 (1987), S. 333-343 
    Details
    ISSN: 1432-0533
    Keywords: Zellweger ; Cytoplasmic striations ; Fatty acids ; Adreno-leukodystrophy ; Neuronal migrations
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Neuropathological examination of three males with cerebro-hepato-renal (Zellweger) syndrome (CHRS) revealed selective neuronal lipidosis and neuroaxonal dystrophy of the dorsal nucleus of Clarke and lateral cuneate nucleus. This lipidotic alteration was visualized as perikaryal or axonal enlargements with cytoplasmic striations. With the light microscope, the striated material was birefringent and resistant to traditional lipid stains; ultrastructurally, it was composed of lipid clefts, lamellae and lamellar-lipid profiles; biochemically, the affected region contained large amounts of cholesterol esterified to very long-chain fatty acids, both saturated and mounounsaturated. This metabolic lesion, though localized to specific sensory neurons, suggests that a more generalized defect in neuronal fatty acid metabolism may be operative in CHRS.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00688256
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  • 10
    Electronic Resource
    Electronic Resource
    Sporadic cerebral amyloid angiopathy with giant cell reaction (1990)
    Springer
    Acta neuropathologica 81 (1990), S. 95-98 
    Details
    ISSN: 1432-0533
    Keywords: Amyloid ; Arteritis ; Congophilic angiopathy ; Intracerebral hemorrhage ; Giant cells
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary We report a case of intracerebral hemorrhages due to sporadic cerebral amyloid angiopathy in a 43-year-old male with a luxuriant giant cell reaction. The amyloid was resistant to potassium permanganate-sulfuric acid oxidation and reacted with an antiserum to synthetic β-protein. The distribution and histologic characteristics of the multinucleated giant cell reaction suggest that it represents a foreign-body reaction rather than giant cell arteritis.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00662644
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