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  • Artikel: DFG Deutsche Nationallizenzen  (2)
  • 1980-1984  (2)
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  • Artikel: DFG Deutsche Nationallizenzen  (2)
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  • 1
    Digitale Medien
    Digitale Medien
    Springer
    European journal of applied physiology 52 (1983), S. 57-61 
    ISSN: 1439-6327
    Schlagwort(e): Sports anemia ; Iron supplements ; Exercise ; Hematologic changes ; Hemoglobin
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Medizin
    Notizen: Summary A young women's exercise/fitness class tested the idea that administration of supplemental iron would prevent “sports anemia” that may develop during exercise and training and improve iron status of exercising females of menstrual age. Fifteen women (aged 18–37) were selected for each of three treatment groups: (1) no supplemental iron; (2) 9 mg·d−1 of Fe; and (3) 18 mg·d−1 of Fe (1 US Recommended Daily Allowance). Women exercised at approximately 85% of maximal heartrate for progressively increasing lengths of time in a jogging program and worked up to 45 min of exercise 4 d·week−1 for 8 weeks. Hematologic analysis was performed in weeks 1, 5, and 8. A significant decline in hemoglobin (Hb) concentration and hematocrit (Hct) was observed at week 5 when all data were examined without regard for iron intake; these red cell indices returned to pre-exercise levels by week 8. Reduction of mean cell hemoglobin concentration (MCHC) indicated that the midpoint decline was not caused by simple hemodilution during exercise. Serum ferritin (SF) concentration changed in parallel with Hb and Hct. Although the midpoint decline in SF was not statistically significant, it ruled out the possibility that turnover of red cell iron was directed to storage. Lowered MCHC and SF suggested lower availability of iron during the synthesis of a new generation of red cells. Few iron treatment effects of magnitude were observed. Iron did not prevent the midpoint decline in Hb concentration. Iron intake did not affect SF, serum iron, transferrin saturation, or final Hb, and Hct. Dietary iron availability thus does not appear to play a role in the phenomenon of “sports anemia”. Temporary alteration of priorities for iron needs during exercise, perhaps for muscle myoglobin, may be responsible for this transitory “anemia”.
    Materialart: Digitale Medien
    Bibliothek Standort Signatur Band/Heft/Jahr Verfügbarkeit
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  • 2
    Digitale Medien
    Digitale Medien
    Springer
    European journal of applied physiology 52 (1984), S. 414-419 
    ISSN: 1439-6327
    Schlagwort(e): Iron deficiency ; Myoglobin ; Endurance training ; Lactic acid ; Hemoglobin
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Medizin
    Notizen: Summary The effects of iron deficiency and endurance training on muscle myoglobin (Mb), body weights, and blood lactic acid concentration were studied in rats. Fifty animals were divided into four groups: anemic trained (AT), normal trained (NT), anemic sedentary (AS), and normal sedentary (NS). Following 5 weeks of dietary control, the mean hemoglobin values for the AT and AS rats were 0.013±0.002 mmol·l−1 (8.7±1.4 g·dl−1) and 0.014±0.003 mmol·l−1 (9.2±1.7 g·dl−1) respectively, and did not significantly change throughout the study. AT and NT rats were run on a motor driven treadmill 4 days/week for 6 weeks up to a pre-established time of 90 min. Following the training, body weights of the AT (157±13 g) and NT (153±13 g) rats were lower than their respective sedentary groups AS (172±9 g) and NS (176±15 g). Resting blood lactic acid concentration following training was lower in both trained groups, AT (3.3±2.0 mM) and NT (2.3±1.9 mM) compared to AS (8.2±2.6 mM) and NS (3.8±1.6 mM). Training increased Mb concentration in hearts of both the anemic and normal trained groups (AT, 0.66±0.13 mg·g−1; NT, 0.95±0.08 mg·g−1) compared to the sedentary groups (AS, 0.44±0.08 mg·g−1; NS, 0.70±0.13 mg·g−1). Only the AT rats showed an increase in skeletal muscle Mb. This study provides evidence that myoglobin may limit aerobic metabolism.
    Materialart: Digitale Medien
    Bibliothek Standort Signatur Band/Heft/Jahr Verfügbarkeit
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