Library

X
feed icon rss

Your email was sent successfully. Check your inbox.

An error occurred while sending the email. Please try again.

Proceed reservation?

Export
Filter
  • 1975-1979
  • 1970-1974  (2)
  • 1974  (2)
Source
Material
Years
  • 1975-1979
  • 1970-1974  (2)
Year
  • 1
    Electronic Resource
    Electronic Resource
    Mutagenicity of chromates in bacteria and its relevance to chromate carcinogenesis (1974)
    [s.l.] : Nature Publishing Group
    Nature 250 (1974), S. 493-495 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] The mechanism of chromate carcinogenicity is not understood. But considerable evidence exists which suggests that chemical carcinogens are also mutagens although the converse is not necessarily true5. We have, therefore, tested the hypothesis that some chromium compounds, including a known ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/250493a0
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Selective stimulation of a cellular immune response by methotrexate (1974)
    Springer
    Inflammation research 4 (1974), S. 113-116 
    Details
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Methotrexate administered to rats undergoing a local graft-versus-host reaction (GvHR) had two quite distinct effects on the lymphoid hypertrophy, that characterizes the GvHR, depending upon the timing of the drug administration: (1) When given after the GvHR is initiated, Methotrexate behaves as a normal cytostatic agent and strongly suppresses the lymphoid hypertrophy. (2) However, when administered before the GvHR is initiated, Methotrexate considerably stimulates the proliferative response of the spleen and draining lymph nodes. Arguments are presented that this latter (stimulatory) effect of Methotrexate may be due to drug action upon a nonimmunological component of the GvHR, contributed by the host, which normally resists/attenuates the aggressive potential of the grafted lymphocytes. The same regimen of Methotrexate had no effect on (1) the aggressive potential of lymphoid cells that passively transfer EAE, or (2) the humoral response to SRBC.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01966820
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
Close ⊗
This website uses cookies and the analysis tool Matomo. More information can be found here...