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  • 1985-1989  (2)
  • 1985  (2)
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  • 1985-1989  (2)
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  • 1
    Electronic Resource
    Electronic Resource
    On the occurrence of the fenestrated vessels in Wallerian degeneration of the peripheral nerve (1985)
    Springer
    Acta neuropathologica 68 (1985), S. 259-262 
    Details
    ISSN: 1432-0533
    Keywords: Peripheral nerve ; Fenestrated endothelium ; Wallerian degeneration
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Ultrastructural studies were made on the distal segments of the mouse phrenic nerve after crush injury. In the control, endoneurium contained only unfenestrated capillaries. In the experiment, from day 2 to day 6, endoneurial capillaries occasionally showed fenestrations with the attenuation of its cytoplasm. At this stage, axonal degeneration and myelin breakdown became evident showing early stage of Wallerian degeneration. In addition, detachment of the neighboring endothelial cells concomitant with the invasion of macrophage was also observed. These findings were previously unobserved changes of the endoneurial endothelium in Wallerian degeneration. The significance of the early occurrence of fenestrae was discussed briefly.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00690205
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Effects of ketamine on the dynorphin levels and the ethylketocyclazocine (EKC) receptor binding in discrete regions of rat brains (1985)
    Springer
    Neurochemical research 10 (1985), S. 945-952 
    Details
    ISSN: 1573-6903
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Intraperitoneal (ip) injection of ketamine increased the concentration of dynorphin in the cortex of rat brain, while decreased it in the septal area. The affinity of ethylketocyclazocine (EKC) receptor binding was decreased in the cortex, but increased in the septal area after pretreating the rats with ketamine. This suggests that the dynorphin neuronal system is stimulated in the cortex and suppressed in the septal area by ketamine. In other 5 brain areas, ketamine had no effect on neither dynorphin concentration nor EKC receptor binding. As dynorphin was reported to produce seizure and spike discharge in the cortex while suppressed the hippocampal EEG of rat brain, it is likely that the dynorphin neuronal system may play at least a part in ketamine induced electrophysiological changes in the brain.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00964631
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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