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  • 1
    Electronic Resource
    Electronic Resource
    Crystallization and preliminary X-ray diffraction studies of the influenza C virus glycoprotein (1996)
    Copenhagen : International Union of Crystallography (IUCr)
    Acta crystallographica 52 (1996), S. 1041-1045 
    Details
    ISSN: 1399-0047
    Source: Crystallography Journals Online : IUCR Backfile Archive 1948-2001
    Topics: Chemistry and Pharmacology , Geosciences , Physics
    Notes: Influenza C virus contains a single surface glycoprotein in its lipid envelope which is the hemagglutinin-esterase-fusion glycoprotein (HEF). HEF binds cell-surface receptors, is a receptor-destroying enzyme (a 9-O-acetylesterase), and mediates the fusion of virus and host cell membranes. A bromelain-released soluble form of HEF has been crystallized. Two different tetragonal forms have been identified from crystals with the same morphology [P1(3)22, a = b = 154.5, c = 414.4 Å, and P41(3)212, a = b = 217.4, c = 421.4 Å]. Both crystal forms share a common packing scheme. Synchrotron data collection and flash cooling of crystals have been used for high-resolution data collection.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1107/S0907444996005021
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  • 2
    Electronic Resource
    Electronic Resource
    Kutane Reaktionen bei Morbus Crohn Vaskulitiden verschiedener Etagen des Hautorgans (1997)
    Springer
    Der Hautarzt 48 (1997), S. 328-331 
    Details
    ISSN: 1432-1173
    Keywords: Schlüsselwörter Morbus Crohn ; Ileitis terminalis ; Kutane Vaskulitis ; Purpura Schönlein-Henoch ; Erythema nodosum ; Key words Crohn disease ; Ileitis terminalis ; Cutaneous vasculitis ; Schönlein-Henoch purpura ; Erythema nodosum
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Summary Two young female patients presented with recurrent erythema and Henoch-Schönlein purpura respectively. In both cases clinical, histological and radiological showed relatively mild, but previously undiagnosed Crohn’s disease as a trigger. We describe the treatment of the underlying disease and the symptomatic treatment of skin lesions. Categories of specific and unspecific skin lesions associated with Crohn’s disease, as described in the literature, are presented. Vasculitic changes of the deep versus the superficial dermal plexus are discussed.
    Notes: Zusammenfassung Wir berichten über 2 junge Patientinnen mit rezidivierendem Erythema nodosum, bzw. Purpura-Schönlein-Henoch unklarer Genese. Bei der klinischen, histologischen und radiologischen Diagnostik zeigte sich in beiden Fällen als Trigger ein relativ mild verlaufender, bisher nicht erkannter Morbus Crohn. Die spezifische Therapie der Grunderkrankung bzw. symptomatische Behandlung der Hauterscheinungen wird beschrieben. Die Einteilung der spezifischen und unspezifischen Crohn-assoziierten Hautsymptome in der Literatur wird dargestellt, und die vaskulitischen Veränderungen des tiefen versus des oberflächlichen dermalen Gefäßplexus werden diskutiert.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s001050050591
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  • 3
    Electronic Resource
    Electronic Resource
    Liver transplantation for the treatment of urea cycle disorders (1998)
    Springer
    Journal of inherited metabolic disease 21 (1998), S. 112-118 
    Details
    ISSN: 1573-2665
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The principal goal of therapy when liver transplantation is used for the treatment of metabolic disease is to correct the metabolic error. By doing so, liver transplantation eliminates the hepatic and peripheral consequences of the error. Inborn errors involving the urea cycle appear on theoretical grounds to be amenable to treatment using liver transplantation and, indeed, published data demonstrate that this approach to therapy can be successful. The purpose of this study is to examine the outcome of liver transplantation done for the indication of urea cycle defects in a large group of patients. The first goal of the study is to determine with certainty that liver transplantation corrects hyperammonaemia and halts the progress of disease. A second goal is to determine the extent of neurological recovery in children previously injured by hyperammonaemia. The final goal is to understand whether the quality of life is improved and medical expense is reduced by transplantation. The study involved a survey of major transplantation centres. Four centres provided data about 16 patients, 14 of whom were alive 11 months to 6 years after transplantation. The results demonstrate that liver transplantation resulted in correction of hyperammonaemia in all patients. The neurological outcome after transplantation correlated closely with the condition prior to transplantation. This population of patients has had relatively few problems in the long term related to the liver transplant itself. The quality of life seems to be much improved, but further study will be needed to confirm this. Limited data involving two patients show a reduction in the cost of care. We conclude from our experience that liver transplantation can be an effective treatment for children with urea cycle defects.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/A:1005317909946
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    Paper (German National Licenses)
    Fulltext
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