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  • 1
    Electronic Resource
    Electronic Resource
    Laser spot scanning in photoelectrochemical systems, relation between spot size and spatial resolution of the photocurrent (1991)
    [S.l.] : American Institute of Physics (AIP)
    Journal of Applied Physics 69 (1991), S. 2324-2327 
    Details
    ISSN: 1089-7550
    Source: AIP Digital Archive
    Topics: Physics
    Notes: Laser spot scanning studies of single-crystalline p-InSe in contact with a neutral aqueous solution reveal a dramatic difference in lateral resolution between the material "as cleaved'' and after platinization by brief dipping in a dilute H2PtCl6 solution. A model is developed to explain these observations, and the resolution is calculated as a function of the diffusion coefficient and the life time of minority carriers, and of the charge transfer rate. The improvement of the resolution is found to be due to the increase of the rate of hydrogen evolution at the illuminated semiconductor by Pt catalyst. The model also provides numerical values for the charge transfer rate in the noncatalyzed and the catalyzed cases.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.348714
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  • 2
    Electronic Resource
    Electronic Resource
    Heterozygous α-antichymotrypsin and PiZ (α1-antitrypsin deficiency (1990)
    Oxford, UK : Blackwell Publishing Ltd
    Allergy 45 (1990), S. 0 
    Details
    ISSN: 1398-9995
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: In a case-control study we compared the prevalence of heterozygous deficiency of two closely related anti-neutrophil protease inhibitors, α1-antitrypsin and α1-antichymotrypsin, in 172 consecutive children with asthma. In a cohort study the clinical spectrum and severity were compared. On the basis of family studies 5/172 (2.9%) were classified as heterozygotes for α1-antichymotrypsin deficiency, a high prevalence compared with that of an unselected adult population (prevalence ratio 4.5 (1.7–11.9), P〈0.005). This finding suggests that the carrier state of this rare allele (prevalence 0.64%) may predispose to asthma in children. Among these heterozygous patients the prevalence of positive RAST tests for foodstuffs was significantly increased (prevalence ratio 4.8 (1.7–13.2). P〈0.005) and 2/5 manifested food allergy with Quincke oedema. Either the PiMZ or SZ phenotype of α1-antitrypsin deficiency was found in 12 (7.0%) of the 172 patients, a prevalence similar to that of a normal population (prevalence ratio 1.3 (0.67–2.6), P= 0.44). However, the asthma was more severe among the Z allele carriers, judged by the number of hospital admissions, compared with the non-Z asthmatic children (mean 2.92 vs. 1.72, P〈0.05). The results indicate that heterozygous deficiency of protease inhibitors directed against neutrophil proteases may affect the severity and clinical spectrum of childhood asthma, and to some degree be predisposing.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1398-9995.1990.tb00483.x
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  • 3
    Electronic Resource
    Electronic Resource
    Hepatocyte inclusions of δ1-antichymotrypsin in a patient with partial deficiency of δ1-antichymotrypsin and chronic liver disease (1990)
    Oxford, UK : Blackwell Publishing Ltd
    Histopathology 16 (1990), S. 0 
    Details
    ISSN: 1365-2559
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: We present a case of chronic liver disease with selective and exclusive hepatocyte endoplasmic reticulum storage of δ1-antichymotrypsin in the form of granules, detected by specific immunohistochemistry at the light microscopy level and corresponding to material found in dilated endoplasmic reticulum of hepatocytes by electron microscopy. The patient had intermediate deficiency of δ1-antichymotrypsin. Thus, the hepatocyte accumulation of δ1-antichymotrypsin may indicate the presence of an export block resembling that of a closely-related protein, namely PiZ δ1-antitrypsin. It is proposed that hepatocyte storage of δ1-antichymotrypsin may be an expression of an inborn error of metabolism bearing the characteristics of endoplasmic reticulum storage diseases such as PiZ δ1-antitrypsin deficiency and hereditary hypofibrinogenaemia.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2559.1990.tb01107.x
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  • 4
    Electronic Resource
    Electronic Resource
    Prognosis after stroke in diabetic patients. A controlled prospective study (1990)
    Springer
    Diabetologia 33 (1990), S. 244-249 
    Details
    ISSN: 1432-0428
    Keywords: Diabetes mellitus ; cerebrovascular disorders ; mortality ; recurrent stroke ; myocardial infarction ; causes of death
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Cohorts of diabetic (n=121) and non-diabetic (n=584) patients were prospectively followed for up to ten years after having suffered from a stroke. All but six of the diabetic patients had Type 2 (non-insulin-dependent) diabetes mellitus. The diabetic patients had more risk factors associated with stroke: heart failure (p〈0.001) and angina pectoris (p〈0.001), than the non-diabetic patients. Neither body mass index nor blood pressure levels differed between the groups at admission. Haematocrit levels were higher in the diabetic group (p〈0.01). The diabetic patients were more commonly afflicted by cerebral embolism and to a lesser extent by transient ischaemic attacks than the nondiabetic patients. When calculated by log-rank tests, the diabetic group had an increased risk of death (p〈0.001), recurrent stroke (p=0.001), and of myocardial infarction (p=0.001) after the initial stroke. Autopsy-verified causes of death between the groups did not differ significantly, although half of all deaths during the period one to six months after stroke were caused by pulmonary embolism in the diabetic group. Thus, diabetes increases the risk of death after a stroke, and it also increases among stroke survivors the risk of recurrent stroke and myocardial infarction.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00404803
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  • 5
    Electronic Resource
    Electronic Resource
    Low levels of coagulation inhibitors in patients withClostridium difficile infection (1992)
    Springer
    Infection 20 (1992), S. 58-60 
    Details
    ISSN: 1439-0973
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung In Seren von Patienten mitClostridium difficile-assoziierter Diarrhoe und Colitis wurden mit kommerziell erhältlichen Antigenassays immunchemisch die Gerinnungsinhibitoren Antithrombin III, Protein C und freies Protein S bestimmt. Im Vergleich zu den Kontrollen hatten die Seren bei allen Patienten mitC. difficile-assoziierter Diarrhoe und Colitis signifikant erniedrigte Inhibitor-Spiegel (Student t-Test). Die Werte für Protein C (Mittelwert ± SD): 0,70±0,30 gegenüber 1,28±0,23, t=6,61, p〈0,001; Antithrombin: 0,70±0,21 gegenüber 0,90±0,17, t=3.12, p〈0,01; freies Protein S: 0,27±0,06 gegenüber 0,37±0,08, t=3,7, p〈0,001. Die Infektion mitC. difficile kann zu einem Verlust an Koagulaseinhibitoren führen und stellt ein Risiko für thromboembolische Komplikationen dar.
    Notes: Summary To investigate levels of coagulation inhibitors in sera from patients withClostridium difficile-associated diarrhoea and colitis, commercially available antigen assays were used for immunochemical determination of antithrombin III, protein C and free protein S. Sera from patients withClostridium difficile-associated diarrhoea and colitis showed significantly lowered levels of all measured inhibitors as compared to controls (Student's t test). Protein C (mean ± SD): 0.70±0.30 vs. 1.28±0.23, t=6.61, p〈0.001; antithrombin: 0.70±0.21 vs. 0.90±0.17, t=3,12, p〈0.01; free protein S: 0.27±0.06 vs. 0.37±0.08, t=3.7, p〈0.001. Infection withC. difficile may lead to loss of coagulation inhibitors and constitutes a risk for thromboembolic complications.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01711063
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