Library

X
feed icon rss

Your email was sent successfully. Check your inbox.

An error occurred while sending the email. Please try again.

Proceed reservation?

Export
  • 1
    Electronic Resource
    Electronic Resource
    Usurpation and nest rebuilding inPolistes riparius: Two ways to reproduce after the loss of the original nest (Hymenoptera: Vespidae) (1989)
    Springer
    Insectes sociaux 36 (1989), S. 116-128 
    Details
    ISSN: 1420-9098
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Description / Table of Contents: Resume Dans une population de la guêpe monogyne,Polistes riparius, le nid est parfois usurpé par une fondatrice étrangère dont le nid a été détruit avant l'apparition des ouvrières. La probabilité de l'usurpation est 0.11–0.23 en phase de pré-émergence. La fondatrice qui n'a pas encore d'ouvrières reconstruit rarement le nid détruit, tandis que celle qui est accompagnée d'ouvrières le reconstruit fréquemment. Par suite, il semble que la fondatrice tente d'usurper ou de prendre possession du nid d'une autre quand son propre nid est détruit avant l'apparition d'ouvrières; au contraire, après leur apparition, elle tente de reconstruire le nid détruit avec ses propres ouvrières. Environ 70% des nids détruits en sous-phase de pré-mâle sont reconstruits, et environ 70% des nids ainsi reconstruits produisent une moyenne de 10–20 imagos reproducteurs, tandis que des nids ordinaires en produisent 60–90. Les usurpatrices détruisent, en moyenne, 80% des œufs et 40% des jeunes larves des fondatrices précédentes pendant 5 jours. Environ 20% des usurpatrices réussit à produire des reproducteurs. Les autres ne produisent aucun imago: la cause de l'échec la plus importante est la destruction de leur couvain par des ouvrières des fondatrices précédentes. On discute l'usurpation des nids établis et la reconstruction du nid détruit du point de vue des tactiques alternatives pour la fondatrice.
    Notes: Summary In a population of the haplometrotic paper waspPolistes riparius, a nest is sometimes usurped by a foundress who has lost its nest before worker emergence. Probability of a nest's being usurped during the pre-emergence stage was 0.11–0.23. Foundresses who did not yet have workers rarely rebuilt destroyed nests, while those with workers frequently did so. Therefore, it seems that a foundress tends to usurp another's nest if her original nest is destroyed before worker emergence, whereas she rebuilds it if her workers have already emerged. About 70% of nests which were destroyed in the pre-male substage were rebuilt, and about 70% of the rebuilt nests produced a mean of 10–20 reproductives of both sexes as compared to 60–90 for non-destroyed nests. On average, usurping foundresses destroyed 80% of the eggs and 40% of the young larvae of the previous queen during 5 days. About 20% of the usurpers succeeded in producing reproductive offspring. Others failed in reproduction. The most important cause of failure was destruction of their broods by workers of the previous foundresses. Usurpation of established nests and nest rebuilding are discussed in terms of alternative tactics of the foundress.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02225907
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Cloning and CO2-dependent expression of the genetic region for encapsulation from Bacillus anthracis (1988)
    Oxford, UK : Blackwell Publishing Ltd
    Molecular microbiology 2 (1988), S. 0 
    Details
    ISSN: 1365-2958
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology , Medicine
    Notes: The capsule of Bacillus anthracis is an important virulence factor consisting of poly-o-glutamic acid. The genetic region required for the encapsulation was cloned in Escherichia coli from the capsule plasmid pTE702, using a selection procedure based on an immunodlffusion assay. The cloned region directed synthesis of the capsule both In E. coli and B. anthracis. Capsule synthesis from these clones, as in the wild type, was dependent upon the presence of CO2. However, encapsulation directed by the cloned fragment was less marked than from pTE702. Another region enhancing capsulation was shown to exist on pTE702. The minimum size of the encapsulation region was defined to within 2.7 kb DNA and shown to be essential for the encapsulation in B. anthracis.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2958.1988.tb00041.x
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Changes in airway responsiveness and β- and α-adrenergic receptors in the lungs of guinea pigs with experimental asthma (1989)
    Oxford, UK : Blackwell Publishing Ltd
    Allergy 44 (1989), S. 0 
    Details
    ISSN: 1398-9995
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: The effects of inhaled allergen on airway responsiveness and on β- and α-1-adrenergic receptors on lung membrane were investigated in guinea pigs. After measuring the respiratory threshold to histamine (RT-HIS), one group of guinea pigs passively sensitized for ovalbumin was challenged by allergen inhalation (challenged group). Measurement of the RT-HIS 24 h following challenge revealed a significant decrease from 687 μg/ml (mean, n= 16) to 407 μg/ml (P 〈 0.05). In addition the RT-HIS 24 h after challenge was also significantly lower in the challenged group than in controls (n= 9, P 〈 0.05). The density of α-adrenergic receptors on the lung membrane of the challenged group was 594 ± 32 (mean ± SE) fmol/mg protein (n= 11) compared with 712 ± 24 fmol/mg protein (n= 9) in the controls, a statistically significant difference (P 〈 0.05). A significant correlation was found between the RT-HIS and density of β-adrenergic receptors. From these results, we concluded that the exaggerated airway responsiveness 24 h after allergen challenge is in part due to a decrease in the density of P-adrenergic receptors. There was no difference in the density of β-1-adrenergic receptors nor a significant correlation between the RT-HIS and the number of α-1-adrenergic receptors in the challenged vs. the control groups.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1398-9995.1989.tb00448.x
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 4
    Electronic Resource
    Electronic Resource
    Molecular cloning and characterization of chromosomal virulence region kcpA of Shigella flexneri (1989)
    Oxford, UK : Blackwell Publishing Ltd
    Molecular microbiology 3 (1989), S. 0 
    Details
    ISSN: 1365-2958
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology , Medicine
    Notes: In Shigella flexneri, in addition to several well-recognized plasmid-borne virulence loci, at least three genetic loci implicated in pathogenesis have been recognized on the chromosome. To understand more about the pathogenesis of bacillary dysentery at a molecular level, the genetically recognized but previously unidentified KcpA region (one of the chromosomal regions near purE) was cloned and sequenced. A single translatable open reading frame encoding a 12310 Dalton protein corresponding to the minicell product was found. Immunofluorescence microscopy, as well as optical and electron microscopic comparison of tissue-cultured cells and guinea-pigs’eyes infected with wild-type or kcpA mutant bacteria, revealed that the kcpA product is required by invading bacteria for spread into adjacent cells.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2958.1989.tb01809.x
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 5
    Electronic Resource
    Electronic Resource
    A dual transcriptional activation system for the 230 kb plasmid genes coding for virulence-associated antigens of Shigella flexneri (1989)
    Oxford, UK : Blackwell Publishing Ltd
    Molecular microbiology 3 (1989), S. 0 
    Details
    ISSN: 1365-2958
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology , Medicine
    Notes: The expression of plasmid-encoded, invasion-related antigens lpa b, c and d of Shigella flexneri was found to be positively regulated at transcriptional level by a 33kD protein produced by the previously defined, virulence-associated Region 1 on the SalI fragment B of the 230 kb invasion plasmid. The gene (designated virB) was identified and its nucleotide sequence determined. No Ipa b or c was produced in the absence of an intact virB gene although lower levels of d were produced. The previously reported regulatory activity of the virF gene some 30 kb distance away was shown to act exclusively through virB. In contrast, the activation of the virG gene necessary for intercellular spread occurred directly by virF without the requirement for virB. This study thus ascribes a critical function to a previously recognized, but functionally undefined, virulence locus on the large invasion plasmid of S. flexneri. The virF gene appears to have a central role in activation of the 230kb plasmid-encoded virulence genes.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2958.1989.tb00210.x
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 6
    Electronic Resource
    Electronic Resource
    The cataract Shionogi mouse, a sister strain of the non-obese diabetic mouse: similar class II but different class I gene products (1988)
    Springer
    Diabetologia 31 (1988), S. 254-258 
    Details
    ISSN: 1432-0428
    Keywords: Major histocompatibility complex ; non-obese diabetic mouse ; non-obese non-diabetic mouse ; cataract Shionogi mouse ; insulin-dependent diabetes ; restriction fragment length polymorphisms
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary We have studied with a series of monoclonal antibodies and restriction fragment analysis the K, D, and class II region of the major histocompatibility complex of the non-obese diabetic mouse in comparison with its sister strains, the non-obese non-diabetic and cataract Shionogi mouse. (1) K region: Monoclonal antibody 31-3-4S (anti-Kd) reacted with splenocytes from non-obese diabetic mice while other anti-K (Kb, Kk, Kq) monoclonals did not react. Splenocytes from non-obese non-diabetic mice reacted with both anti-Kb and Kk monoclonals while splenocytes from cataract Shionogi mice reacted with anti-Kd and Kk monoclonals. Both sister strains, therefore, differ from the non-obese diabetic and other known mice strains by monoclonal analysis of H-2K. (2) D region: Splenocytes from both non-obese diabetic and non-obese non-diabetic mice reacted with monoclonal antibody 28-14-8S (anti-Db) while splenocytes from cataract Shionogi mice did not react with any anti-D monoclonal antibody tested. (3a) Class II region (non-obese diabetic and non-obese non-diabetic mice): Three of 11 monoclonal antibodies to class II molecules reacted with splenocytes of the non-obese diabetic mouse. The 3 reacting monoclonals have I-Ak primary specificities though additional anti-I-Ak monoclonal antibodies were negative. Among these monoclonals, 39B and 40A reacted with the non-obese diabetic mouse but not with the non-obese non-diabetic mouse, while 10-2-16 reacted with non-obese diabetic, non-obese non-diabetic and cataract Shionogi mice. Monoclonal MKD6 (anti-I-Ad) reacted with non-obese non-diabetic but not non-obese diabetic mice. In crosses of non-obese diabetic with non-obese non-diabetic mice, splenocytes from all diabetic backcrosses studied (6/6) were positive with monoclonal 40A but negative with MKD6 indicating that the major histocompatibility complex of non-obese non-diabetic mice is not diabetogenic and confirming major histocompatibility complex-linkage of the diabetogenic gene in this additional cross. (3b) Class II region (cataract Shionogi mice): Utilising 11 anti-class II monoclonal antibodies the pattern of reactivity of the cataract Shionogi mouse was identical to the non-obese diabetic mouse. Splenocytes from both non-obese diabetic and cataract Shionogi mice fail to express I-E (no reaction with monoclonal 14-4-4). In addition, using an I-A alpha probe with restriction endonuclease HindIII or I-A beta probe with BamHI, the restriction fragment length polymorphism pattern of the cataract Shionogi mouse was identical to the non-obese diabetic mouse. In summary, the cataract Shionogi mouse appears to have a similar I-A region to the non-obese diabetic mouse but differs at both the K and D region. With the hypothesis that the unique I-A beta of the non-obese diabetic mouse is diabetogenic, the cataract Shionogi mouse should provide a new strain for further characterisation of diabetogenic genes of the non-obese diabetic mouse since it is similar at I-A, fails to express I-E, but differs at both class I loci.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00290594
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
Close ⊗
This website uses cookies and the analysis tool Matomo. More information can be found here...