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Color Stability of Astaxanthin Pigmented Rainbow Trout Under Various Packaging Conditions (1984)

CHEN, HUEI-MEI ; MEYERS, S. P. ; HARDY, R. W. ; [et al.]

Oxford, UK : Blackwell Publishing Ltd

Journal of food science 49 (1984), S. 0

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ISSN: 1750-3841

Source: Blackwell Publishing Journal Backfiles 1879-2005

Topics: Agriculture, Forestry, Horticulture, Fishery, Domestic Science, Nutrition , Process Engineering, Biotechnology, Nutrition Technology

Notes: Astaxanthin-rich oil extract from Louisiana crawfish by-product imparted a salmon-red coloration to the flesh and integument of rainbow trout on a diet containing 45 mg astaxanthin/kg feed. Carotenoids deposited in fish flesh were stable (ca 90% retention) in air-packed samples at 1 - 2°C. Oxygen-evacuated and CO2-enriched packaging gave adverse color stability. Lowest pigment levels were in frozen samples, with 53% of the absorbed pigment degraded in 14 days. Visual color scores were consistent with quantitative pigment analyses (r = 0.77). Higher pigment contents were observed in air-packed (1 - 2°C) samples, possibly due to astacene or other breakdown products of astaxanthin. Maximal TBA values were observed in air-packed (1 - 2°C) samples. The effect of tissue enzyme homogenate on fading of trout flesh is discussed.

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1111/j.1365-2621.1984.tb14985.x

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Analysis of spermatogenesis in Drosophila melanogaster bearing deletions for Y-chromosome fertility genes (1981)

Hardy, R. W. ; Tokuyasu, K. T. ; Lindsley, D. L.

Springer

Chromosoma 83 (1981), S. 593-617

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ISSN: 1432-0886

Source: Springer Online Journal Archives 1860-2000

Topics: Biology , Medicine

Notes: Abstract The effects on spermatogenesis of a series of contiguous non-overlapping Y-chromosome deficiencies were examined using both the light and electron microscope. The deficiencies were constructed by combining elements of different X-Y translocations; they subdivide the Y into seven segments, six of which are required for male fertility (four in the long arm and two in the short arm). Spermatogensis was examined from the primary spermatocyte through to the formation of mature sperm and the earliest departures from normal development identified. Two deficiencies result in the absence of the same structure from the axoneme of the sperm tail-the dynein-containing outer arm extending from the A subtubule of the peripheral doublet; they also result in the absence from primary spermatocyte nuclei of aggregates of tubuli in one case and reticular material in the other. A third deficiency causes the appearance in the primary spermatocyte of the crystals characteristic of X0 males and the irregular distribution during meiosis of nuclear and cytoplasmic elements to the spermatids. The fourth deficiency results in the misalignment of the developing axoneme with the mitochondrial derivatives and is first detectable in the onion nebenkern stage of the spermatid. Finally for two deficiencies the first abnormalties detected were during later stages and comprise a syndrome found in most of the steriles. We attribute this phenotype to the indirect effects of earlier lesions.