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  • 1960-1964  (6)
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Material
Years
Year
  • 1
    Electronic Resource
    Electronic Resource
    The myocardium and hemodynamics in cardiac hyperfunction and insufficiency (1963)
    Springer
    Bulletin of experimental biology and medicine 54 (1963), S. 1092-1096 
    Details
    ISSN: 1573-8221
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary In cases withvitium cordis only a part of work done by the ventricle is transformed into its external work. All the work performed by the ventricle may be designated as total work, whereas the ratio of external and total work is designated as mechanical efficiency of the ventricle. The study of the dynamics of external, total work and of the mechanical efficiency of the left ventricle in the process of progressive aortic stenosis development demonstrated that in the given experimental vitium cordis the energy losses for overcoming the blood flow resistance in the area of stenosis lead to a 4-fold reduction in the mechanical efficiency of the left ventricle (as compared to the normal). Suggestions are presented on determination of total work and of mechanical efficiency in other forms of vitium cordis; it is also suggested that reduction of the efficiency of the heart in cardiac insufficiency depends not only on disturbed myocardial metabolism, but also on the drop of mechanical efficiency of the heart.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00784603
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Changes in the intramural nervous apparatus in compensatory cardiac hypertrophy (1962)
    Springer
    Bulletin of experimental biology and medicine 52 (1962), S. 1213-1216 
    Details
    ISSN: 1573-8221
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Changes in the intramural nerve ganglia and in the myocardium caused by experimentally induced compensatory cardiac hyperfunction followed a definite pattern. During the first stage there was an edema of the neurones, various stages of diffuse lysis of the Nissl substance, and vacuolization of the processes of the nerve cell. The second stage was one of relatively stable hyperfunction, during which the edema disappeared from most of the neurones. A small proportion of the neurones were irreversibly damaged, and died.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00787290
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Changes in the intensity of protein synthesis in the myocardium during compensatory hyperfunction of the heart (1961)
    Springer
    Bulletin of experimental biology and medicine 50 (1961), S. 678-680 
    Details
    ISSN: 1573-8221
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Compensatory hyperfunction of the heart in vitium cordis, hypertension and other diseases of the circulatory system connected with the cardiac overstrain of long duration represents the main factor in preservation of normal hemodynamic indices and clinical compensation. The rate of protein synthesis in the myocardium was studied under conditions of compensatory cardiac hyperfunction provoked by experimentally induced vitium cordis. As established, rapid development of hypertrophy during the first emergency stage of compensatory hyperfunction is accompanied by an intensified rate of protein synthesis (it about doubles). In the second stage of relatively stable hyperfunction the rate of protein synthesis is normal, while at the third stage — characterized by cardiosclerosis and progressive exhaustion —it is reduced 2–2.5 times. This depression of the normal renovation process of the protein myocardial structures may bring about a change in the contractile proteins of the heart, thus playing an important role in the development of cardiosclerosis and cardiac insufficiency.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00796034
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    Paper (German National Licenses)
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  • 4
    Electronic Resource
    Electronic Resource
    The myocardium and the hemodynamics during cardiac hyperfunction and insufficiency. Report 2. Lowering of the resistance of the vascular field as an economic compensatory factor in diseases of the circulatory system (1964)
    Springer
    Bulletin of experimental biology and medicine 55 (1964), S. 26-29 
    Details
    ISSN: 1573-8221
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary During gradual reproduction of experimental aortic stenosis in dogs there occurs a considerable reduction of the general vascular resistance. This shift is clearly observed not only in stenosis of the aorta, it is an important compensatory factor maintaining the normal cardiac output in economic use of the myocardial reserves. A drop in vascular resistance during intensive cardiac hyperfunction may be due to the reflux from the receptors of the left ventricle to the arteriolar musculature.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00800194
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    Paper (German National Licenses)
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  • 5
    Electronic Resource
    Electronic Resource
    Increased effect of extrarenal pressor factors following removal of kidneys (1961)
    Springer
    Bulletin of experimental biology and medicine 50 (1961), S. 913-916 
    Details
    ISSN: 1573-8221
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Experiments on rabbits have shown that the pressor effect of adrenalin and pituitrin increases considerably 48 hours after removal of both kidneys. This result confirms the authors' opinion that hypertension due to nephrectomy may be caused by an increased effect of extrarenal pressor factors without any change in their amount.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00797478
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    Paper (German National Licenses)
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  • 6
    Electronic Resource
    Electronic Resource
    Effect of nucleosides, strophanthin, and combinations of these factors on the development of fatigue in the myocardium (1964)
    Springer
    Bulletin of experimental biology and medicine 57 (1964), S. 547-551 
    Details
    ISSN: 1573-8221
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary A study was made of the effect produced by nucleosides (uridine, inosine and bases included into their composition—uracil and hypoxanthine) on the development of fatigue in the myocardium. The latter process was induced in acute experimental conditions by repeated overloading of the left cardiac ventricle by complete occlusion of the ascending aorta for 30 sec. The interval between the occlusions was 5 min. The maximal systolic pressure observed during aortic compression decreased with each subsequent compression, notwithstanding the unchanged or rising endodiastolic pressure. The dynamics of reduction of the maximal level of cardiac contractile function characterizes the process of fatigue. Administration of uridine, inosine and hypoxanthine following the fifth compression inhibits the further development of the fatigue process; the same effect is produced by strophanthin. Administration of a combination of uridine, uracil inosine, hypoxanthine and strophanthin eliminated the fatigue phenomena and increased the maximal level of the cardiac contractile function to a greater degree and for a longer time than separate administration of the same agents.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00782314
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    Paper (German National Licenses)
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