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  • Amezinium  (1)
  • Bladder outlet obstruction  (1)
  • Catecholamine  (1)
  • 1
    Electronic Resource
    Electronic Resource
    Catecholamine metabolism in the vas deferens and the adrenal gland with special reference to the central catecholamine-depleted state (1992)
    Springer
    Cellular and molecular life sciences 48 (1992), S. 667-671 
    Details
    ISSN: 1420-9071
    Keywords: Catecholamine ; vas deferens ; adrenal gland ; 6-hydroxydopamine ; alpha-methyl-para-tyrosine
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Experiments were carried out to elucidate the role of central catecholamines in regulating catecholamine metabolism in the vas deferens and adrenal gland of the rat. Rats were injected intracerebroventricularly (i.c.v.) with either vehicle or 6-hydroxydopamine (6-OHDA). Groups of animals pretreated with vehicle or 6-OHDA (i.c.v.) were injected intraperitoneally (i.p.) with alpha-methyl-para-tyrosine (AMT), a tyrosine hydroxylase inhibitor. Catecholamine turnover rates were estimated by determining norepinephrine or epinephrine content after administrating AMT. Central norepinephrine and dopamine contents decreased significantly (p〈0.05) after treatment with 6-OHDA and AMT. The norepinephrine content of the vas deferens of rats pretreated with 6-OHDA was markedly reduced (p〈0.001) after administration of AMT, whereas that of the vehicle-treated rats remained unchanged. Administration of 6-OHDA had no effect on the norepinephrine or epinephrine content of the adrenal gland. The present results indicate that central monoaminergic neurons have an inhibitory effect on the adrenergic neurons of the vas deferens. In contrast, this inhibitory regulation does not appear to be exerted on the adrenal glands.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02118315
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Morphometric analysis of symptomatic benign prostatic hyperplasia with and without bladder outlet obstruction (2000)
    Springer
    Urological research 28 (2000), S. 29-32 
    Details
    ISSN: 1434-0879
    Keywords: Key words Prostatic hyperplasia ; Morphometry ; Bladder outlet obstruction ; Smooth muscle
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract To investigate the histological features of benign prostatic hyperplasia (BPH) with and without bladder outlet obstruction (BOO), 32 patients with symptomatic BPH, 22 with BOO and ten without BOO, were studied. The area density of smooth muscle, fibrous tissue, glandular epithelium, and glandular lumen in BPH adenoma resected by transurethral surgery was estimated using morphometric analysis. No differences were found in the area density of stromal and glandular components between the two groups. In the glandular component, neither the epithelium nor lumen differed between the two groups. However, the area density of smooth muscle in the obstructed group showed a decrease compared to that in the unobstructed group (P 〈 0.01). We conclude that a decreased proportion of smooth muscle in BPH adenoma may play an important role in BOO.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s002400050006
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Direct effect of amezinium on rabbit urethra: Effect of estrogen and progesterone treatment (1996)
    Springer
    International urogynecology journal 7 (1996), S. 325-330 
    Details
    ISSN: 1433-3023
    Keywords: Amezinium ; Estrogen ; Progesterone ; Rabbit urethra
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The effect of amezinium, a new antihypotensive agent, and hormonal treatment on the female rabbit urethra was investigated. Cumulative dose responses were obtained for amezinium and norepinephrine on strips of muscle from the urethras of ovariectomized female rabbits by means of the tissuebath system. Amezinium enhanced the response to electrical field stimulation and showed a direct contractile response on the urethra. These responses were only about 20% of the maximum norepinephrine response. The contractile response to amezinium was completely blocked by prazosin. When rabbits were pretreated with estrogen, with or without progesterone, for 4 weeks, the response to amezinium increased to 40% of the maximum norepinephrine response. Although amezinium enhances muscle contractile responses to electrical stimulation, this effect is strong when amezinium is used alone; concurrent estrogen treatment improves the effects of amezinium.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01901108
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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