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  • Cathepsin B  (1)
  • Glibenclamide  (1)
  • 1
    Digitale Medien
    Digitale Medien
    Cathepsin-B activity in otosclerosis (1983)
    Springer
    European archives of oto-rhino-laryngology and head & neck 238 (1983), S. 123-125 
    Details
    ISSN: 1434-4726
    Schlagwort(e): Otosclerosis ; Stapes footplate ; Cathepsin B
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Medizin
    Notizen: Summary Cathepsin-B activity was determined fluorimetrically in the otosclerotic stapes footplate, the stapes superstructure, normal temporal cortical bone, and os frontale osteoma. Measurements with a synthetic substrate made determinations in individual samples possible. The cathepsin-B activity in the otosclerotic stapes footplate was one order of magnitude higher than that of the superstructure, which was not affected by the disease. The cortical bone and the superstructure displayed similar activities, as did os frontale osteoma and otosclerosis. The high lysosomal proteinase activity appears to be closely connected to the otosclerotic bone resorption process.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1007/BF00454303
    Bibliothek Standort Signatur Band/Heft/Jahr Verfügbarkeit
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  • 2
    Digitale Medien
    Digitale Medien
    Involvement of glibenclamide-sensitive potassium channels in vasorelaxation by cochlear nerve stimulation (1997)
    Springer
    European archives of oto-rhino-laryngology and head & neck 254 (1997), S. S6 
    Details
    ISSN: 1434-4726
    Schlagwort(e): Cochlea ; Field stimulation ; Vascular endothelium ; Vasorelaxation ; Glibenclamide
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Medizin
    Notizen: Abstract Rabbit aortic rings relaxed with an increase in cyclic guanosine monophosphate and cyclic adenosine monophosphate content in response to exposure to organ fluid of isolated cochleas of the guinea pig following field stimulation (50 Hz, 80 V, 0.2 ms). Relaxations were blocked by 30 μMN G-nitro-l-arginine methyl ester added to the vessel rings. This inhibitory effect was reversed by 3 MMl-arginine. Removal of the vascular endothelium also blocked the relaxation response. Glibenclamide attenuated vasorelaxation in a concentration-dependent manner. We conclude that cochlear nerve stimulation induces an endothelium-dependent vasorelaxation involving activation of adenosine triphosphate-sensitive potassium channels.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1007/BF02439710
    Bibliothek Standort Signatur Band/Heft/Jahr Verfügbarkeit
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    Artikel (Nationallizenzen)
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