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  • 1
    Electronic Resource
    Electronic Resource
    Urinary tamm-horsfall protein as a marker of renal transplant function (1994)
    Springer
    Urological research 22 (1994), S. 131-136 
    Details
    ISSN: 1434-0879
    Keywords: Kidney transplantation ; Tamm-Horsfall protein ; Acute tubular necrosis ; Rejection episodes ; Cytomegalovirus infection
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract In a total of 428 urine samples collected from 15 patients aged between 23 and 60 years after cadaveric kidney transplantation during a postoperative hospital stay, Tamm-Horsfall protein (THP) was quantitatively determined using theELIAS SYNELISA-THP immunoassay. All patients were treated with azathioprine, cyclosporine, prednisolone, given an intraoperative high-dose single antilymphocyte globulin bolus and discharged with functioning grafts. In clinically uncomplicated courses, even after immediate transplant function, the recovery of graft function took on average 7 days. Thereafter the urinary THP excretion was relatively stable and amounted, on average, to 14.5 ± 4.9 mg/24 h (i.e. was at the lower limit of normal urinary THP excretion). In cases of delayed onset of graft function of undetermined origin accompanied by extremely reduced urinary THP excretion, the functional recovery, whether spontaneous or brought about by treatment, was characterized by a continuous increase in urinary THP excretion. In connection with interstitial rejections urinary THP excretion seems not to be a recommendable diagnostic parameter. Daily measurement of urinary THP is, however, suitable for monitoring the functional state of transplanted kidneys.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00571838
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  • 2
    Electronic Resource
    Electronic Resource
    Die Phagocytoseaktivität von permanenten Fibroblasten (Monocyten), Rattenalveolarmakrophagen und menschlichen Entzündungsmakrophagen unter der Wirkung von Glucocorticoiden (1967)
    Springer
    Clinical and experimental medicine 144 (1967), S. 335-352 
    Details
    ISSN: 1591-9528
    Keywords: Glucocorticoides ; phagocytosis ; macrophages ; fibroblastes ; dust exposition ; Glucocorticoide ; Phagocytose ; Makrophagen ; Fibroblasten ; Staubexposition
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung Glucocorticoide hemmen die Phagocytoseaktivität von permanenten Fibroblasten und Makrophagen in vitro. In der Kultur gezüchtete Monocyten gewöhnten sich bei einer Corticoiddauerbehandlung an das veränderte Milieu, ihre anfänglich gehemmte Phagocytoseaktivität nahm wieder zu. Makrophagen aus Rattenlungen sowie menschliche Entzündungsmakrophagen zeigten nach Corticoidbehandlung eine deutliche Abnahme ihrer Phagocytoseaktivität, die bei den Rattenmakrophagen von der Dauer der Behandlung und der Dosis abhängig war. Bei Bestaubung von corticoidbehandelten Ratten konnte ebenfalls eine von der Behandlungsdauer abhängige Abnahme der Phagocytoseaktivität im Alveolarraum beobachtet werden. Bei Kurzbehandlung zeigte sich eine bimodale Wirkung des applizierten Glucocorticoids durch gesteigerte Makrophagenemigration in den Alveolarraum.
    Notes: Summary Glucocorticoides inhibit the phagocytosis activity of permanent fibroblasts and macrophages in vitro. Monocytes raised in culture become accustomed to the changed medium during a continuous corticoide treatment; their initially inhibited phagocytosis activity increased again. Macrophages from rat lungs as well as human inflammation macrophages showed after corticoide treatment a marked decrease of their phagocytosis activity dependent, in case of rat macrophages on the duration of treatment and dose. During dusting of rats treated with corticoides, there could also be observed a decrease of the phagocytosis activity in the alveolar region dependent on the duration of treatment. In case of a short treatment there was a bimodal effect of the applied glucocorticoides showed by increased macrophage emigration into the alveolar region.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02043239
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    Paper (German National Licenses)
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