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  • 1
    ISSN: 1432-0630
    Keywords: PACS: 62.20 Qp; 68.55.Jk; 81.65.Lp
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics , Physics
    Notes: Abstract. This work presents the surface treatment of commercial titanium alloy by means of a Nd:YAG (1.064 μm) laser in the presence of nitrogen. The treated surface was characterised by using scanning electron microscopy, atomic force microscopy, X-ray diffraction, secondary ion mass spectrometry, UV-visible spectrophotometry and microindentation. Experimental results show the formation of a nitrogen-rich layer, 500 nm thick, with a surface morphology characterised by the presence of polygonal structures which suggest that melting occurred under the action of the laser. The δ phase of titanium nitride was identified, in addition to a nitrogen in α-titanium solid solution. The reflectance spectrum of the yellow-golden sample is similar to that of deposited titanium nitride thin films. The hardness of the treated surface was measured by microindentation and found to be 14 GPa, a value three times higher than that of the titanium alloy. In scratch tests the nitrided layer detached at a load of 0.9 N.
    Type of Medium: Electronic Resource
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  • 2
    ISSN: 1435-232X
    Keywords: Key words Nonsense mutation ; Complement component 7 ; Deficiency
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Deficiency of the seventh component of complement (C7D) is frequently associated with recurrent neisserial infections. We report in the present study the genetic basis for C7D in a Spanish family. We used exon-specific polymerase chain reaction (PCR)/single-strand conformation polymorphism (SSCP) analysis as a screening step for mutations, followed by direct sequencing of the target exon. The mutation in the proband was a homozygous G-to-T transversion at nucleotide 1957, the first nucleotide of the codon GAG for Glu-631, leading to a stop codon TAG (E631X). Our result provides further evidence that the molecular pathogenesis of C7D is heterogeneous.
    Type of Medium: Electronic Resource
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