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  • 1
    Electronic Resource
    Electronic Resource
    Specificity of androgen resistance inMus caroli kidney (1988)
    Springer
    Biochemical genetics 26 (1988), S. 705-716 
    Details
    ISSN: 1573-4927
    Keywords: Mus caroli ; Mus musculus ; androgen ; kidney ; submandibular gland
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology
    Notes: Abstract Androgen controls the expression of β-glucuronidase and several other proteins in the kidney of the standard laboratory mouse,Mus musculus. Other species within the genusMus exhibit a variety of response patterns for kidney β-glucuronidase and other markers of androgen action. We have investigated the mechanism of androgen action inM. caroli, aMus species that does not produce β-glucuronidase in response to testosterone. The failure of testosterone to induce β-glucuronidase inM. caroli females cannot be overcome by treatment with dihydrotestosterone, with pharmacological doses of testosterone propionate or dihydrotestosterone propionate, or with a variety of potent androgen analogues. All of these compounds induce kidney β-glucuronidase inM. musculus females and kidney ornithine decarboxylase, submandibular gland renin, and submandibular gland epidermal growth factor in bothM. caroli andM. musculus females. Furthermore, kidney androgen receptor proteins fromM. caroli andM. musculus animals have the same sedimentation characteristics on sucrose density gradients. These data indicate that androgen resistance inM. caroli is not due to deficient 5α-reductase or aberrant hormone metabolism producing suboptimal levels of functional androgen and is not caused by a defective androgen receptor. They suggest that the resistance of β-glucuronidase inM. caroli kidney to induction by androgen occurs at the level of the β-glucuronidase gene.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02395517
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Specificity of androgen resistance inMus caroli kidney (1988)
    Springer
    Biochemical genetics 26 (1988), S. 705-716 
    Details
    ISSN: 1573-4927
    Keywords: Mus caroli ; Mus musculus ; androgen ; kidney ; submandibular gland
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology
    Notes: Abstract Androgen controls the expression of β-glucuronidase and several other proteins in the kidney of the standard laboratory mouse,Mus musculus. Other species within the genusMus exhibit a variety of response patterns for kidney β-glucuronidase and other markers of androgen action. We have investigated the mechanism of androgen action inM. caroli, aMus species that does not produce β-glucuronidase in response to testosterone. The failure of testosterone to induce β-glucuronidase inM. caroli females cannot be overcome by treatment with dihydrotestosterone, with pharmacological doses of testosterone propionate or dihydrotestosterone propionate, or with a variety of potent androgen analogues. All of these compounds induce kidney β-glucuronidase inM. musculus females and kidney ornithine decarboxylase, submandibular gland renin, and submandibular gland epidermal growth factor in bothM. caroli andM. musculus females. Furthermore, kidney androgen receptor proteins fromM. caroli andM. musculus animals have the same sedimentation characteristics on sucrose density gradients. These data indicate that androgen resistance inM. caroli is not due to deficient 5α-reductase or aberrant hormone metabolism producing suboptimal levels of functional androgen and is not caused by a defective androgen receptor. They suggest that the resistance of β-glucuronidase inM. caroli kidney to induction by androgen occurs at the level of the β-glucuronidase gene.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00553870
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Short-term effects of a catastrophic beaver dam collapse on a stream fish community (1991)
    Springer
    Environmental biology of fishes 31 (1991), S. 123-129 
    Details
    ISSN: 1573-5133
    Keywords: Benthic insects ; Colonization dynamics ; Disturbance ; Floods ; Landscape ecology ; Minnesota
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Synopsis We examined the short-term effects of the natural catastrophic collapse of a beaver dam on downstream benthic insect density and fish community structure in a headwater tributary of the Mississippi River. The catastrophic collapse of the dam and ensuing flash flood resulted in a dramatic (〈 90%) decrease in benthic insect density in riffle and pool habitats. Sixty days after collapse of the dam, insect densities in riffles were 62% of pre-collapse densities. Insect recolonization of pools was slower than for riffles; 60 days after collapse of the dam insect densities in pools were 8% of pre-collapse levels. Collapse of the beaver dam altered the structure of the downstream fish community by causing a short-term (2–4 days) influx of pond species, resulting in a brief increase in species richness and abundance. Fish species richness and abundance then decreased for 4–60 days to levels below those prior to the collapse.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00001012
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
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