ISSN:
1432-1238
Keywords:
Hypokalemia
;
Potassium shift
;
Catecholamine release
;
Head trauma
;
Beta-2-stimulation
Source:
Springer Online Journal Archives 1860-2000
Topics:
Medicine
Notes:
Abstract A sudden decrease of serum potassium below 2.5 mmol/l carries the risk of dangerous arrhythmias and requires immediate replacement therapy [6]. We refer to a patient with a brain stem compression after head injury, who developed a profound hypokalemia (K+=1.2 mmol/l) with life-threatening arrhythmias, probably due to a catecholamine induced intracellular potassium shift (beta-2-stimulation). Only by aggressive potassium replacement up to 80 mmol/h (610 mmol/16h) could potassium levels be increased and cardiac arrhythmias terminated. Although replacement therapy was stopped when the serum K+-level increased to 2.4 mmol/l, 3.5 h later the patient became hyperkalemic (8.1 mmol/l). This was probably due to a secondary shift of potassium from intra-to extracellular space. In patients with severe head trauma and the potential risk of excessive catecholamine release special attention must be paid to changes in potassium balance.
Type of Medium:
Electronic Resource
URL:
http://dx.doi.org/10.1007/BF01701479
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