Abstract
We describe a case of incomplete locked-in syndrome (LIS) due to basilar artery thrombosis, in which MRI showed a complete, sharply demarcated infarct at the pontomedullary junction. This supports experimental data showing that the lower reticular formation is not critical for the maintenance of consciousness. To our knowledge, this is the first reported case of ischaemic pontomedullary transection with LIS.
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References
Lindeberg R, Freytag E (1970) Brainstem lesions characteristic of traumatic hyperextension of the head. Arch Pathol 90: 509–515
Britt RH, Herrick MK, Hamilton RD (1977) Traumatic locked-in syndrome. Ann Neurol 1: 590–592
Virgile RS (1984) Locked-in syndrome. Case and literature review. Clin Neurol Neurosurg 86: 275–279
Patterson JR, Grabois M (1986) Locked-in syndrome: a review of 139 cases. Stroke 17: 758–764
Latronico N, Candiani A (1987) Brainstem herpes virus encephalitis (letter). Lancet II: 690–691
Bauer G, Gerstenbrand F, Rumpl E (1979) Variables of the locked-in syndrome. J Neurol 221: 77–91
Plum F, Posner JB (1982) The pathologic physiology of signs and symptoms of coma. In: Plum F, Posner JB (eds) The diagnosis of stupor and coma, 3rd edn. Davis, Philadelphia, pp 13–15
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Latronico, N., Tansini, A., Gualandi, G.F. et al. Ischaemic pontomedullary transection with incomplete locked-in syndrome. Neuroradiology 35, 332–334 (1993). https://doi.org/10.1007/BF00588361
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DOI: https://doi.org/10.1007/BF00588361