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Changes in the intramural nervous apparatus in compensatory cardiac hypertrophy

  • Morphology and Pathomorphology
  • Published:
Bulletin of Experimental Biology and Medicine Aims and scope

Summary

Changes in the intramural nerve ganglia and in the myocardium caused by experimentally induced compensatory cardiac hyperfunction followed a definite pattern. During the first stage there was an edema of the neurones, various stages of diffuse lysis of the Nissl substance, and vacuolization of the processes of the nerve cell. The second stage was one of relatively stable hyperfunction, during which the edema disappeared from most of the neurones. A small proportion of the neurones were irreversibly damaged, and died.

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Literature Cited

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Authors and Affiliations

  1. B. I. Lavrent'ev Laboratory of Neurohistology, Institute of Normal and Pathological Physiology, AMN SSSR, Moscow

    E. M. Krokhina, F. Z. Meerson & A. L. Mikoélyan

  2. Laboratory of Physiology and Pathophysiology of Cardiac Activity, Institute of Normal and Pathological Physiology, AMN SSSR, Moscow

    E. M. Krokhina, F. Z. Meerson & A. L. Mikoélyan

Authors
  1. E. M. Krokhina
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  2. F. Z. Meerson
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  3. A. L. Mikoélyan
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Krokhina, E.M., Meerson, F.Z. & Mikoélyan, A.L. Changes in the intramural nervous apparatus in compensatory cardiac hypertrophy. Bull Exp Biol Med 52, 1213–1216 (1962). https://doi.org/10.1007/BF00787290

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  • DOI: https://doi.org/10.1007/BF00787290

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