Summary
Feeding under conditions of reduced oxygen supply prompted an increase in serum, arterial and brain tissue levels of thiobarbituric acid (TBA)-reacting substances. These observations indicated the possibility that hypoxia might be one of the factors predisposing to the accumulation of lipid peroxide.
Similar content being viewed by others
References
W.C. Hueper, Archs Path.38, 173 (1944).
P. Astrup and K. Kjeldsen, Med. Clins N. Am.58, 323 (1973).
I. Lorenzen and P. Helin, Acta path. microbiol. scand.69, (dy1967).
J. Glavind, S. Hartman, J. Clemmesen, K.E. Jessen and S. Dan, Acta path. microbiol. scand.30, 1 (1952).
K. Yagi, Biochem. Med.15, 212 (1976).
H. Ohkawa, N. Ohishi and K. Yagi, Analyt. Biochem.95, 351 (1979).
Author information
Authors and Affiliations
Rights and permissions
About this article
Cite this article
Yoshikawa, T., Furukawa, Y., Wakamatsu, Y. et al. The increase of thiobarbituric acid reacting substances in rats with experimental chronic hypoxia. Experientia 38, 312–313 (1982). https://doi.org/10.1007/BF01949361
Published:
Issue Date:
DOI: https://doi.org/10.1007/BF01949361