Central catecholaminergic control of ACTH secretion
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Hypothermic responses to infection are inhibited by α<inf>2</inf>- adrenoceptor agonists with possible clinical implications
2009, British Journal of AnaesthesiaCitation Excerpt :Infusion of either the α2-agonists or the antagonists failed to affect basal or LPS-stimulated plasma corticosterone levels. This result was unexpected, since several previous studies have demonstrated a role for α2-adrenoceptors in the regulation of the HPA axis.37–42 Hence α2-agonists and antagonists have been shown to, respectively, attenuate and augment basal and stimulated adrenocorticotrophic hormone (ACTH) and corticosterone responses.37 38 43
Progressive dysregulation of autonomic and HPA axis functions in HIV-1 clade C infection in South India
2008, PsychoneuroendocrinologyCitation Excerpt :This sequential neuroendocrine regulatory pathway thus prevents the deleterious effects of stress on the brain (Sapolsky et al., 1986; Sapolsky, 1996). Although the exact mechanisms involving the disconnect between the attenuated response of ACTH, and hyper response of cortisol to star tracing challenge in HIV-1C infected individuals is not clearly understood at present, it may be suggested that it is a reflection of an attenuated noradrenergic response to MSTCT found in this study, since the release of ACTH from the pituitary is also stimulated by the noradrenergic system (Takao et al., 1988). Similar findings from other studies also suggest that autonomic response to life events regulate the HPA axis activity and that integration of the central autonomic system with CNS is essential for the activation of the HPA axis (Malarkey et al., 1994).
WAY-100635 inhibits 8-OH-DPAT-stimulated oxytocin, ACTH and corticosterone, but not prolactin secretion
1998, European Journal of PharmacologyMonoaminergic regulation of neuroendocrine function and its modification by cocaine
1994, Frontiers in Neuroendocrinology