Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
Alterations of peroxisomal function in ischemia-reperfusion injury of rat kidney
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Scutellarin acts on the AR-NOX axis to remediate oxidative stress injury in a mouse model of cerebral ischemia/reperfusion injury
2022, PhytomedicineCitation Excerpt :GSTK1 plays an important role in maintaining mitochondrial function and homeostasis in the pathogenesis of type 2 diabetes (Deng et al., 2020). Studies have reported that acyl-CoA oxidase is sensitive to ischaemia-reperfusion injury (Gulati et al.,1993), peroxisome-mediated oxidative stress mediates protection against renal dysfunction in hypertensive diabetic rats (Dhaunsi et al.,2010), and peroxisomal Acox1 has anti-inflammatory and antiaging properties. Inflammation occurs throughout the whole pathological process of cerebral ischaemia, and the correlation between AR and inflammation has also been fully verified (Singh et al., 2021).
Beyond rare disorders: A new era for peroxisomal pathophysiology
2022, Molecular CellCitation Excerpt :For more discussion on which directions can benefit from such research, see Box 1. For example, while it is clear that peroxisomes are affected in both their number and activity during cases of ischemia-reperfusion injury (Gulati et al., 1993; Singh and Gulati, 1995) and that activation of peroxisomal fatty acid oxidation helps ameliorate the damage in such cases, (Liepinsh et al., 2013) still very little has been studied regarding peroxisomes and this central disease state (Delille et al., 2006). Various degenerative diseases have also not been well characterized in connection to peroxisomes, including muscular dystrophy, pulmonary emphysema and rheumatoid arthritis (Berger et al., 2016; Masters and Crane, 1995).
The Role of Peroxisome Proliferator-Activated Receptor γ Coactivator 1α (PGC-1α) in Kidney Disease
2018, Seminars in NephrologyCitation Excerpt :During reperfusion, oxygen tension will increase, which in theory should restore FAO immediately. However, studies have indicated that FAO enzymes remain suppressed during the reperfusion phase16 and lipid droplets accumulate.17 In endotoxin-induced sepsis AKI models, PGC-1α level is decreased and its level correlate with the degree of renal impairment.17,18
Effect of trimetazidine on the nucleotide profile in rat kidney with ischemia-reperfusion injury
2006, European Journal of Pharmaceutical SciencesAlcohol-induced Membrane Damage
2004, Comprehensive Handbook of Alcohol Related Pathology