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  • Articles: DFG German National Licenses  (4)
  • Electronic Resource  (4)
  • 1990-1994  (2)
  • 1985-1989  (2)
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  • Articles: DFG German National Licenses  (4)
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  • Electronic Resource  (4)
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  • 1
    Electronic Resource
    Electronic Resource
    Influence of air pollutants and nutrient deficiency on D-1 protein content and photosynthesis in young spruce trees (1992)
    Oxford, UK : Blackwell Publishing Ltd
    Physiologia plantarum 85 (1992), S. 0 
    Details
    ISSN: 1399-3054
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology
    Notes: Long-term fumigation with different mixtures of air pollutants, such as O3, NO2 and SO2, in combination with potassium deficiency in 4-year-old spruce trees shows clearly that the photosynthetic apparatus reacts very sensitively to environmental stress. Even when no change in photosynthetic oxygen evolution of the leaves was measurable, the content of D-1 protein in the reaction center of photosystem II already increased significantly at low stress conditions. With increasing concentrations of pollutants, the content of D-1 protein decreased considerably. This pattern was observed with all combinations of gases, although they differed in effectiveness. The most potent effect on the content of D-1 protein was found with a combined fumigation of SO2 plus NO2. Obviously, the ratio between synthesis and degradation of the D-1 protein is influenced by stress factors. However, the content of D-1 protein was not strongly correlated with photosynthetic oxygen evolution in whole needles nor with electron transport rate in isolated thylakoids. Under controlled potassium deficiency, effects similar to those observed upon ozone fumigation developed. The results suggest unspecific stress answers for the D-1 content and the photosynthetic reactions rather than specific responses.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1399-3054.1992.tb04762.x
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Effects of brofaremine (CGP 11 305A), a short-acting, reversible, and selective inhibitor of MAO-A on sleep, nocturnal penile tumescence and nocturnal hormonal secretion in three healthy volunteers (1987)
    Springer
    Psychopharmacology 92 (1987), S. 110-114 
    Details
    ISSN: 1432-2072
    Keywords: Brofaremine ; Sleep ; Nocturnal penile tumescence ; Cortisol ; Human growth hormone ; Prolactin ; Testosterone ; LH ; FSH
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The effects of brofaremine (CGP 11 305A), a short-acting, reversible and selective inhibitor of MAO-A, on sleep, nocturnal penile tumescence (NPT) and hormonal secretion during the night were studied during a long-term trial. Three healthy males underwent sleep-EEG and NPT recordings during consecutive nights (1) under placebo, (2) under stepwise increasing dosages of brofaremine and (3) under placebo after withdrawal. Hormone profiles were sampled during selected nights to analyze the plasma concentration of cortisol, HGH, prolactin, testosterone, LH and FSH. REM sleep was suppressed markedly under 150 mg brofaremine, while stages 1 and 2 increased. In comparison to the effect of irreversible MAOIs the REM suppression was shorter and did not persist after withdrawal. A decrease of the plasma concentration of the drug coincided with a return of sleep variables to baseline values. A REM rebound occurred after withdrawal of brofaremine. REM sleep and NPT showed a dissociation; NPT variables did not follow the decrease of REM sleep. The effects of REM parameters are correlated with the dosage and the plasma concentration of the substance. Intraindividually, a decrease in secretion of HGH was observed throughout the trial. No marked changes were found in the other endocrinological variables.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00215489
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    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Flumazenil exerts intrinsic activity on sleep EEG and nocturnal hormone secretion in normal controls (1994)
    Springer
    Psychopharmacology 113 (1994), S. 334-338 
    Details
    ISSN: 1432-2072
    Keywords: Sleep EEG ; Cortisol ; Growth hormone ; Flumazenil ; Midazolam
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The physiological function of benzodiazepine (BDZ) receptors includes regulation of sleep and neuroendocrine activity. Most of the pharmacological effects of BDZ are blocked by flumazenil. However, recent neurological and behavioral studies suggest that flumazenil has its own central intrinsic activity. This issue was addressed in a study of the sleep EEG and the nocturnal secretion of growth hormone and cortisol in ten normal male controls, who were given flumazenil either alone or in combination with the BDZ agonist midazolam, placebo and midazolam alone. Flumazenil prompted an increase in sleep onset latency, a decrease in slow wave sleep and an increase in wakefulness. Plasma cortisol concentrations after flumazenil administration were lower than after midazolam. Both flumazenil and midazolam decreased nocturnal growth hormone secretion. After simultaneous application of both BDZ receptor ligands the growth hormone blunting was amplified. Our study demonstrates that at the level of the sleep EEG and neuroendocrine activity flumazenil is capable of exerting both agonistic and inverse agonistic or antagonistic effects.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02245206
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    Paper (German National Licenses)
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  • 4
    Electronic Resource
    Electronic Resource
    Examination and treatment of sleep-related painful erections—a case report (1989)
    Springer
    Archives of sexual behavior 18 (1989), S. 263-267 
    Details
    ISSN: 1573-2800
    Keywords: sleep-related painful erections ; sleep-EEG ; NPT ; clozapine
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine , Psychology
    Notes: Abstract The case of a patient with sleep-related painful erections is described. Insomnia and a slight depressive syndrome occurred along with a long history of this disorder. No physical abnormality was found. At a baseline recording of sleep electroencephalography (EEG) and nocturnal penile tumescence (NPT), a disturbed sleep pattern and impaired NPT were recorded. Attempts to treat the disorder with diazepam, amitriptyline, trimipramine, and biperidene did not prompt a stable improvement of the disorder, but a dosage of 25 mg clozapine was sufficient to achieve normalized sleep architecture, remission of the depressive symptomatology, and normalization of NPT. It is likely that marked sedation is the mode of action of clozapine.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01543200
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    Paper (German National Licenses)
    Fulltext
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